Role of extracellular HSP72 in acute stress-induced potentiation of innate immunity in active rats. (Q54536327)

31 May 2018

title

Role of extracellular HSP72 in acute stress-induced potentiation of innate immunity in active rats. (English)

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31 May 2018

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31 May 2018

author name string

J Campisi

series ordinal

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31 May 2018

language of work or name

English

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publication date

13 September 2002

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Journal of Applied Physiology

published in

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volume

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issue

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page(s)

43-52

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Mechanisms of inflammation and leukocyte activation

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Necrotic but not apoptotic cell death releases heat shock proteins, which deliver a partial maturation signal to dendritic cells and activate the NF-kappa B pathway

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Tumor necrosis factor-alpha expression induced by anti-YopB antibodies coincides with protection against Yersinia enterocolitica infection in mice

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21 January 2018

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Proinflammatory cytokines

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Activity wheel running blunts increased plasma adrenocorticotrophin (ACTH) after footshock and cage-switch stress

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Acute stressor exposure facilitates innate immunity more in physically active than in sedentary rats.

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Cutting edge: heat shock protein (HSP) 60 activates the innate immune response: CD14 is an essential receptor for HSP60 activation of mononuclear cells

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Intracellular monocyte and serum cytokine expression is modulated by exhausting exercise and cold exposure

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Stress-induced susceptibility to bacterial infection during cutaneous wound healing

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10.1152/JAPPLPHYSIOL.00681.2002

21 January 2018

Identifiers

DOI

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31 May 2018

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31 May 2018