Human umbilical cord blood mononuclear cell-conditioned media inhibits hypoxic-induced apoptosis in human coronary artery endothelial cells and cardiac myocytes by activation of the survival protein Akt. (Q54455328)

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English	Human umbilical cord blood mononuclear cell-conditioned media inhibits hypoxic-induced apoptosis in human coronary artery endothelial cells and cardiac myocytes by activation of the survival protein Akt.	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/23336598

Human umbilical cord blood mononuclear cell-conditioned media inhibits hypoxic-induced apoptosis in human coronary artery endothelial cells and cardiac myocytes by activation of the survival protein Akt. (English)

1 reference

Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/23336598

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Paul R. Sanberg

2

object named as

Paul R Sanberg

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Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/23336598

author name string

Hua Jin

1

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Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/23336598

Robert J Henning

3

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Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/23336598

publication date

16 January 2013

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Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/23336598

Cell Transplantation

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Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/23336598

22

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Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/23336598

9

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Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/23336598

1637-1650

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Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/23336598

Protein kinase activation and myocardial ischemia/reperfusion injury

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The Akt activation inhibitor TCN-P inhibits Akt phosphorylation by binding to the PH domain of Akt and blocking its recruitment to the plasma membrane

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Factors secreted by mesenchymal stem cells and endothelial progenitor cells have complementary effects on angiogenesis in vitro

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Akt Phosphorylation of BAD Couples Survival Signals to the Cell-Intrinsic Death Machinery

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Activation of nitric oxide synthase in endothelial cells by Akt-dependent phosphorylation

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Regulation of the inflammatory response in cardiac repair

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Regulation of endothelium-derived nitric oxide production by the protein kinase Akt

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Human umbilical cord blood mononuclear cells decrease fibrosis and increase cardiac function in cardiomyopathy.

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Human umbilical cord blood progenitor cells are attracted to infarcted myocardium and significantly reduce myocardial infarction size

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Human cord blood cells and myocardial infarction: effect of dose and route of administration on infarct size

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Human umbilical cord blood mononuclear cells activate the survival protein Akt in cardiac myocytes and endothelial cells that limits apoptosis and necrosis during hypoxia.

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H9c2 cell line is a valuable in vitro model to study the drug metabolizing enzymes in the heart

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Identifiers

10.3727/096368912X661427

1 reference

Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/23336598

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

30 May 2018

http://europepmc.org/abstract/MED/23336598

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