The anti-apoptotic gene BCL2A1 is a novel transcriptional target of PU.1. (Q54438848)

29 May 2018

title

The anti-apoptotic gene BCL2A1 is a novel transcriptional target of PU.1. (English)

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29 May 2018

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Torsten Haferlach

object named as

T Haferlach

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M Jenal

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J Batliner

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V A Reddy

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A Tobler

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M F Fey

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B E Torbett

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publication date

4 March 2010

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published in

Leukemia

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volume

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issue

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page(s)

1073-1076

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PU.1 is a lineage-specific regulator of tyrosine phosphatase CD45.

cites work

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https://api.crossref.org/works/10.1038%2FLEU.2010.26

Transcription factors in myeloid development: balancing differentiation with transformation.

21 January 2018

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https://api.crossref.org/works/10.1038%2FLEU.2010.26

Disruption of differentiation in human cancer: AML shows the way.

21 January 2018

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https://api.crossref.org/works/10.1038%2FLEU.2010.26

The BCL-2 protein family: opposing activities that mediate cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2010.26

Bcl-XL expression correlates with primary macrophage differentiation, activation of functional competence, and survival and results from synergistic transcriptional activation by Ets2 and PU.1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2010.26

Accelerated neutrophil apoptosis in mice lacking A1-a, a subtype of the bcl-2-related A1 gene.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2010.26

PU.1, a novel caspase-3 substrate, partially contributes to chemotherapeutic agents-induced apoptosis in leukemic cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2010.26

PU.1 induces apoptosis in myeloma cells through direct transactivation of TRAIL

21 January 2018

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https://api.crossref.org/works/10.1038%2FLEU.2010.26

Retinoic acid-induced apoptosis in leukemia cells is mediated by paracrine action of tumor-selective death ligand TRAIL.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2010.26

Potential autoregulation of transcription factor PU.1 by an upstream regulatory element

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2010.26

The prosurvival Bcl-2 homolog Bfl-1/A1 is a direct transcriptional target of NF-kappaB that blocks TNFalpha-induced apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2010.26

Retinoic acid determines life span of leukemic cells by inducing antagonistic apoptosis-regulatory programs

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2010.26

ATRA resolves the differentiation block in t(15;17) acute myeloid leukemia by restoring PU.1 expression

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2010.26

Cytoprotective response of A1, a Bcl-2 homologue expressed in mature human neutrophils and promyelocytic HL-60 cells, to oxidant stress-induced cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2010.26

The Ets2 transcription factor inhibits apoptosis induced by colony-stimulating factor 1 deprivation of macrophages through a Bcl-xL-dependent mechanism.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2010.26

Upregulation of Bfl-1/A1 in leukemia cells undergoing differentiation by all-trans retinoic acid treatment attenuates chemotherapeutic agent-induced apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2010.26

PU.1 binding to the p53 family of tumor suppressors impairs their transcriptional activity.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2010.26

Spi-1/PU.1 participates in erythroleukemogenesis by inhibiting apoptosis in cooperation with Epo signaling and by blocking erythroid differentiation

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2010.26

inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1038/LEU.2010.26

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29 May 2018

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29 May 2018