Innate stimulation of B1a cells enhances the autoreactive IgM repertoire in the NOD mouse: implications for type 1 diabetes. (Q54321119)

27 May 2018

title

Innate stimulation of B1a cells enhances the autoreactive IgM repertoire in the NOD mouse: implications for type 1 diabetes. (English)

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27 May 2018

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27 May 2018

Carlos Penha-Gonçalves

series ordinal

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27 May 2018

N Duarte

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J Côrte-Real

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27 May 2018

publication date

1 March 2012

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27 May 2018

published in

Diabetologia

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27 May 2018

volume

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issue

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27 May 2018

page(s)

1761-1772

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Incidence trends for childhood type 1 diabetes in Europe during 1989-2003 and predicted new cases 2005-20: a multicentre prospective registration study

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B lymphocytes are essential for the initiation of T cell-mediated autoimmune diabetes: analysis of a new "speed congenic" stock of NOD.Ig mu null mice

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Marginal zone and B1 B cells unite in the early response against T-independent blood-borne particulate antigens

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Inflammatory tendencies and overproduction of IL-17 in the colon of young NOD mice are counteracted with diet change

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Anergy and not clonal ignorance determines the fate of B cells that recognize a physiological autoantigen

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Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

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Characterization of antibody responses to endogenous and exogenous antigen in the nonobese diabetic mouse

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Early expression of antiinsulin autoantibodies of humans and the NOD mouse: evidence for early determination of subsequent diabetes

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B-1a B cells that link the innate and adaptive immune responses are lacking in the absence of the spleen

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Autoimmunity triggers in the NOD mouse: a role for natural auto-antibody reactivities in type 1 diabetes

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CD22 x Siglec-G double-deficient mice have massively increased B1 cell numbers and develop systemic autoimmunity

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Human CD5 promotes B-cell survival through stimulation of autocrine IL-10 production

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Peritoneal B cells govern the outcome of diabetes in non-obese diabetic mice

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I-Ag7-mediated antigen presentation by B lymphocytes is critical in overcoming a checkpoint in T cell tolerance to islet beta cells of nonobese diabetic mice

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Irf4 is a positional and functional candidate gene for the control of serum IgM levels in the mouse

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B lymphocytes are critical antigen-presenting cells for the initiation of T cell-mediated autoimmune diabetes in nonobese diabetic mice.

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B cell specificity contributes to the outcome of diabetes in nonobese diabetic mice

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10.1007/S00125-012-2498-0

Identifiers

DOI

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27 May 2018

1 reference

27 May 2018