Phosphatidylinositol 3-kinase and NF-kappaB regulate motility of invasive MDA-MB-231 human breast cancer cells by the secretion of urokinase-type plasminogen activator (Q43786822)

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scientific article published on 31 October 2001

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English	Phosphatidylinositol 3-kinase and NF-kappaB regulate motility of invasive MDA-MB-231 human breast cancer cells by the secretion of urokinase-type plasminogen activator	scientific article published on 31 October 2001

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scholarly article

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Europe PubMed Central

PubMed publication ID

26 November 2017

Phosphatidylinositol 3-kinase and NF-kappaB regulate motility of invasive MDA-MB-231 human breast cancer cells by the secretion of urokinase-type plasminogen activator (English)

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Europe PubMed Central

PubMed publication ID

26 November 2017

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author name string

Daniel Sliva

1

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Europe PubMed Central

PubMed publication ID

26 November 2017

Maria T Rizzo

2

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Europe PubMed Central

PubMed publication ID

26 November 2017

Denis English

3

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Europe PubMed Central

PubMed publication ID

26 November 2017

language of work or name

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publication date

31 October 2001

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Europe PubMed Central

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26 November 2017

Journal of Biological Chemistry

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Europe PubMed Central

PubMed publication ID

26 November 2017

277

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Europe PubMed Central

PubMed publication ID

26 November 2017

5

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Europe PubMed Central

PubMed publication ID

26 November 2017

3150-3157

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

Regulation of chemotaxis by the platelet-derived growth factor receptor-beta

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An 11-amino acid sequence from c-met initiates epithelial chemotaxis via phosphatidylinositol 3-kinase and phospholipase C.

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21 January 2018

The lipid products of phosphoinositide 3-kinase increase cell motility through protein kinase C.

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Signalling through the lipid products of phosphoinositide-3-OH kinase

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The role of phosphoinositide 3-kinase lipid products in cell function

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Using structure to define the function of phosphoinositide 3‐kinase family members

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Xid-like immunodeficiency in mice with disruption of the p85alpha subunit of phosphoinositide 3-kinase.

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Impaired B cell development and proliferation in absence of phosphoinositide 3-kinase p85alpha.

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Aberrant nuclear factor-kappaB/Rel expression and the pathogenesis of breast cancer

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Activation of phosphatidylinositol 3-kinase in response to interleukin-1 leads to phosphorylation and activation of the NF-kappaB p65/RelA subunit

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uPA, uPAR, PAI-I: Key intersection of proteolytic, adhesive and chemotacfic highways?

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The chemotactic action of urokinase on smooth muscle cells is dependent on its kringle domain. Characterization of interactions and contribution to chemotaxis

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Tip60 interacts with human interleukin-9 receptor alpha-chain

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Wortmannin inactivates phosphoinositide 3-kinase by covalent modification of Lys-802, a residue involved in the phosphate transfer reaction

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Mitogenic signaling of insulin-like growth factor I in MCF-7 human breast cancer cells requires phosphatidylinositol 3-kinase and is independent of mitogen-activated protein kinase.

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The activated insulin-like growth factor I receptor induces depolarization in breast epithelial cells characterized by actin filament disassembly and tyrosine dephosphorylation of FAK, Cas, and paxillin.

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Novel functions of phosphatidylinositol 3-kinase in terminally differentiated cells

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Specific Requirement for the p85-p110α Phosphatidylinositol 3-Kinase during Epidermal Growth Factor-stimulated Actin Nucleation in Breast Cancer Cells

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21 January 2018

Interleukin 8-stimulated phosphatidylinositol-3-kinase activity regulates the migration of human neutrophils independent of extracellular signal-regulated kinase and p38 mitogen-activated protein kinases

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21 January 2018

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21 January 2018

Phosphatidylinositol 3-kinase in interleukin 1 signaling. Physical interaction with the interleukin 1 receptor and requirement in NFkappaB and AP-1 activation

1 reference

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21 January 2018

Requirement of phosphatidylinositol 3-kinase activity for bradykinin stimulation of NF-kappaB activation in cultured human epithelial cells

1 reference

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The interferon-inducible p202 protein as a modulator of transcription: inhibition of NF-kappa B, c-Fos, and c-Jun activities

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Transforming growth factor-beta1 enhances the invasiveness of human MDA-MB-231 breast cancer cells by up-regulating urokinase activity.

1 reference

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21 January 2018

Identifiers

10.1074/JBC.M109579200

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

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