Hippocampal neurons exhibit both persistent Ca2+ influx and impairment of Ca2+ sequestration/extrusion mechanisms following excitotoxic glutamate exposure (Q43540595)

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scientific article published in March 2001

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English	Hippocampal neurons exhibit both persistent Ca2+ influx and impairment of Ca2+ sequestration/extrusion mechanisms following excitotoxic glutamate exposure	scientific article published in March 2001

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

24 November 2017

Hippocampal neurons exhibit both persistent Ca2+ influx and impairment of Ca2+ sequestration/extrusion mechanisms following excitotoxic glutamate exposure (English)

1 reference

Europe PubMed Central

PubMed publication ID

24 November 2017

1 reference

based on heuristic

inferred from title

author name string

Limbrick DD Jr

1

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Europe PubMed Central

PubMed publication ID

24 November 2017

Pal S

2

1 reference

Europe PubMed Central

PubMed publication ID

24 November 2017

DeLorenzo RJ

3

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Europe PubMed Central

PubMed publication ID

24 November 2017

language of work or name

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publication date

1 March 2001

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Europe PubMed Central

PubMed publication ID

24 November 2017

1 reference

Europe PubMed Central

PubMed publication ID

24 November 2017

894

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Europe PubMed Central

PubMed publication ID

24 November 2017

1

1 reference

Europe PubMed Central

PubMed publication ID

24 November 2017

56-67

1 reference

Europe PubMed Central

PubMed publication ID

24 November 2017

Neuronal calcium signaling

1 reference

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Sodium/calcium exchange: its physiological implications

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Intracellular calcium levels during the period of delayed excitotoxicity

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The pharmacology of intracellular Ca(2+)-release channels

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Glutamate receptor-induced 45Ca2+ accumulation in cortical cell culture correlates with subsequent neuronal degeneration

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On the origin of a sustained increase in cytosolic Ca2+ concentration after a toxic glutamate treatment of the nerve cell culture

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Glutamate impairs neuronal calcium extrusion while reducing sodium gradient

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Inability to restore resting intracellular calcium levels as an early indicator of delayed neuronal cell death

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The control of neuronal Ca2+ homeostasis

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NMDA receptor-dependent excitotoxicity: the role of intracellular Ca2+ release

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Dantrolene-Na (Dantrium) blocks induction of long-term potentiation in hippocampal slices

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Induction of spontaneous recurrent epileptiform discharges causes long-term changes in intracellular calcium homeostatic mechanisms

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In vitro status epilepticus causes sustained elevation of intracellular calcium levels in hippocampal neurons

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Molecular mechanisms of calcium-dependent excitotoxicity

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Measurement of Intracellular Free Zinc in Living Cortical Neurons: Routes of Entry

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Neurotoxic activation of glutamate receptors induces an extended neuronal depolarization in cultured hippocampal neurons

1 reference

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7 January 2021

Secondary Ca2+ overload indicates early neuronal injury which precedes staining with viability indicators.

1 reference

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Alterations in calcium-mediated signal transduction after traumatic injury of cortical neurons

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7 January 2021

Identifiers

10.1016/S0006-8993(00)03303-5

1 reference

Europe PubMed Central

PubMed publication ID

24 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

24 November 2017

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