Differential regulation of [Ca2+]i oscillations in mouse pancreatic islets by glucose, alpha-ketoisocaproic acid, glyceraldehyde and glycolytic intermediates (Q42496693)

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English	Differential regulation of [Ca2+]i oscillations in mouse pancreatic islets by glucose, alpha-ketoisocaproic acid, glyceraldehyde and glycolytic intermediates	scientific article

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

Differential regulation of [Ca2+]i oscillations in mouse pancreatic islets by glucose, alpha-ketoisocaproic acid, glyceraldehyde and glycolytic intermediates (English)

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

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author name string

S Lenzen

1

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Europe PubMed Central

PubMed publication ID

1 November 2017

M Lerch

2

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Europe PubMed Central

PubMed publication ID

1 November 2017

T Peckmann

3

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Europe PubMed Central

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1 November 2017

M Tiedge

4

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Europe PubMed Central

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1 November 2017

publication date

1 September 2000

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Europe PubMed Central

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1 November 2017

Biochimica et Biophysica Acta

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1 November 2017

1523

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Europe PubMed Central

PubMed publication ID

1 November 2017

1

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Europe PubMed Central

PubMed publication ID

1 November 2017

65-72

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Europe PubMed Central

PubMed publication ID

1 November 2017

Cytoplasmic Ca2+ oscillations in pancreatic beta-cells.

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Periodic patterns in biochemical reactions

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Pulsatile insulin release and electrical activity from single ob/ob mouse islets of Langerhans

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Widespread synchronous [Ca2+]i oscillations due to bursting electrical activity in single pancreatic islets

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Origin of slow and fast oscillations of Ca2+ in mouse pancreatic islets

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Cell photodamage, a potential hazard when measuring cytoplasmic Ca2+ with fura-2

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A decrease of both [Ca2+]e and [H+]e produces cell damage in the perfused rat heart

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Does glyceraldehyde enter pancreatic islet metabolism via both the triokinase and the glyceraldehyde phosphate dehydrogenase reactions? A study of these enzymes in islets

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Effects of pyruvate, L-lactate, and 3-phenylpyruvate on function of ob/ob mouse pancreatic islets: insulin secretion in relation to 45Ca2+ uptake and metabolism

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Insulinotropic action of methyl pyruvate: secretory, cationic, and biosynthetic aspects

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Effects of L-leucine and alpha-ketoisocaproic acid upon insulin secretion and metabolism of isolated pancreatic islets

1 reference

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2-oxocarboxylic acids and function of pancreatic islets in obese-hyperglycaemic mice. Insulin secretion in relation to 45Ca uptake and metabolism

1 reference

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Glucoreceptor mechanisms and the control of insulin release and biosynthesis

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Signal recognition by pancreatic B-cells

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Activation of stimulus-secretion coupling in pancreatic beta-cells by specific products of glucose metabolism. Evidence for privileged signaling by glycolysis.

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Hexose metabolism in pancreatic islets. Glucose-induced and Ca(2+)-dependent activation of FAD-glycerophosphate dehydrogenase

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Calcium activation of mitochondrial glycerol phosphate dehydrogenase restudied

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Role of mitochondrial calcium transport in the control of substrate oxidation

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ATP-sensitive K+ channels and insulin secretion: their role in health and disease.

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Uptake and release of Ca2+ by the endoplasmic reticulum contribute to the oscillations of the cytosolic Ca2+ concentration triggered by Ca2+ influx in the electrically excitable pancreatic B-cell

1 reference

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Regulation of insulin secretion by energy metabolism in pancreatic B-cell mitochondria. Studies with a non-metabolizable leucine analogue

1 reference

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2-ketoglutarate generation in pancreatic B-cell mitochondria regulates insulin secretory action of amino acids and 2-keto acids

1 reference

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Glycine transformation of Ca2+ oscillations into a sustained increase parallels potentiation of insulin release

1 reference

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Amino acid-induced [Ca2+]i oscillations in single mouse pancreatic islets of Langerhans

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inferred from DOI database lookup

Identifiers

10.1016/S0304-4165(00)00100-8

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

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