Transcription factors ETF, E2F, and SP-1 are involved in cytokine-independent proliferation of murine hepatocytes. (Q42478950)

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title

Transcription factors ETF, E2F, and SP-1 are involved in cytokine-independent proliferation of murine hepatocytes (English)

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Reinhard Guthke

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Wolfgang Schmidt-Heck

object named as

Wolfgang Schmidt-Heck

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Rolf Gebhardt

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Irmgard Merfort

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Sebastian Zellmer

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Patricio Godoy

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Honglei Weng

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Christoph Meyer

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Thomas Lehmann

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Titus Sparna

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Wiebke Schormann

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Jens Timmer

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Fritz von Weizsäcker

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Petra A Thürmann

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Steven Dooley

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language of work or name

English

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publication date

26 October 2010

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volume

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issue

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page(s)

2127-2136

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Liver regeneration after partial hepatectomy: critical analysis of mechanistic dilemmas

14 January 2020

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Temporal and functional profile of the transcriptional regulatory network in the early regenerative response to partial hepatectomy in the rat

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Enhanced matrix degradation after withdrawal of TGF-beta1 triggers hepatocytes from apoptosis to proliferation and regeneration

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E2F1 modulates p38 MAPK phosphorylation via transcriptional regulation of ASK1 and Wip1.

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Restoration of liver mass after injury requires proliferative and not embryonic transcriptional patterns

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Role of ERK, p38 and PI3-kinase in EGF receptor-mediated mitogenic signalling in cultured rat hepatocytes: requirement for sustained ERK activation

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The mitogen-activated protein kinase kinase/extracellular signal-regulated kinase cascade activation is a key signalling pathway involved in the regulation of G(1) phase progression in proliferating hepatocytes

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Transcription of the MAT2A gene, coding for methionine adenosyltransferase, is up-regulated by E2F and Sp1 at a chromatin level during proliferation of liver cells

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Embryotrophic factor-3 from human oviductal cells enhances proliferation, suppresses apoptosis and stimulates the expression of the beta1 subunit of sodium-potassium ATPase in mouse embryos.

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Sibling rivalry in the E2F family

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Balancing the decision of cell proliferation and cell fate

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Sp1 coordinately regulates de novo lipogenesis and proliferation in cancer cells

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Sp1: emerging roles--beyond constitutive activation of TATA-less housekeeping genes

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Mechanisms responsible for carbon tetrachloride-induced perturbation of mitochondrial calcium homeostasis.

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Differential inhibitory effects of carbon tetrachloride on the hepatic plasma membrane, mitochondrial and endoplasmic reticular calcium transport systems: implications to hepatotoxicity

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Calcium, calmodulin and cell cycle progression

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Adenosine administration accelerates progression of the cell cycle during rat liver regeneration induced by one-third hepatectomy

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