Release of preformed Fas ligand in soluble form is the major factor for activation-induced death of Jurkat T cells (Q41146738)

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scientific article published on December 1996

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English	Release of preformed Fas ligand in soluble form is the major factor for activation-induced death of Jurkat T cells	scientific article published on December 1996

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instance of

scholarly article

1 reference

stated in

Europe PubMed Central

PubMed publication ID

9014814

retrieved

27 September 2017

title

Release of preformed Fas ligand in soluble form is the major factor for activation-induced death of Jurkat T cells (English)

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Europe PubMed Central

PubMed publication ID

9014814

retrieved

27 September 2017

author name string

Martínez-Lorenzo MJ

series ordinal

1

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Europe PubMed Central

PubMed publication ID

9014814

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27 September 2017

Alava MA

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2

1 reference

stated in

Europe PubMed Central

PubMed publication ID

9014814

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27 September 2017

Anel A

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3

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stated in

Europe PubMed Central

PubMed publication ID

9014814

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27 September 2017

Piñeiro A

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4

1 reference

stated in

Europe PubMed Central

PubMed publication ID

9014814

retrieved

27 September 2017

Naval J

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5

1 reference

stated in

Europe PubMed Central

PubMed publication ID

9014814

retrieved

27 September 2017

language of work or name

English

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based on heuristic

inferred from title

publication date

1 December 1996

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stated in

Europe PubMed Central

PubMed publication ID

9014814

retrieved

27 September 2017

published in

Immunology

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stated in

Europe PubMed Central

PubMed publication ID

9014814

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27 September 2017

volume

89

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Europe PubMed Central

PubMed publication ID

9014814

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27 September 2017

issue

4

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Europe PubMed Central

PubMed publication ID

9014814

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27 September 2017

page(s)

511-517

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Europe PubMed Central

PubMed publication ID

9014814

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27 September 2017

cites work

Transferrin receptors recycle to cis and middle as well as trans Golgi cisternae in Ig-secreting myeloma cells

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

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inferred from PubMed ID database lookup

Role of oxidative damage and IL-1 beta-converting enzyme-like proteases in Fas-based cytotoxicity exerted by effector T cells

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

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Cytolysis by Ca-permeable transmembrane channels. Pore formation causes extensive DNA degradation and cell lysis

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

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inferred from PubMed ID database lookup

A cell-killing monoclonal antibody (anti-Fas) to a cell surface antigen co-downregulated with the receptor of tumor necrosis factor

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PubMed

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https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

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inferred from PubMed ID database lookup

Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

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inferred from PubMed ID database lookup

Increased intracellular cyclic AMP inhibits inositol phospholipid hydrolysis induced by perturbation of the T cell receptor/CD3 complex but not by G-protein stimulation. Association with protein kinase A-mediated phosphorylation of phospholipase C-g

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

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inferred from PubMed ID database lookup

Induction of apoptosis by monoclonal antibody anti-APO-1 class switch variants is dependent on cross-linking of APO-1 cell surface antigens

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Fas involvement in Ca(2+)-independent T cell-mediated cytotoxicity

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PubMed

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https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

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Mature T cells of autoimmune lpr/lpr mice have a defect in antigen-stimulated suicide

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PubMed

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https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

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Molecular cloning and expression of the Fas ligand, a novel member of the tumor necrosis factor family

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Purification and characterization of the Fas-ligand that induces apoptosis

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Generalized lymphoproliferative disease in mice, caused by a point mutation in the Fas ligand

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PubMed

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https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Fas-based lymphocyte-mediated cytotoxicity against syngeneic activated lymphocytes: a regulatory pathway?

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

T cell receptor-induced Fas ligand expression in cytotoxic T lymphocyte clones is blocked by protein tyrosine kinase inhibitors and cyclosporin A

1 reference

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Fas ligand mediates activation-induced cell death in human T lymphocytes

1 reference

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Autocrine T-cell suicide mediated by APO-1/(Fas/CD95)

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PubMed

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https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Cell-autonomous Fas (CD95)/Fas-ligand interaction mediates activation-induced apoptosis in T-cell hybridomas.

1 reference

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Fas(CD95)/FasL interactions required for programmed cell death after T-cell activation.

1 reference

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

TCR/CD3 coupling to Fas-based cytotoxicity

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

The Fas death factor

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

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inferred from PubMed ID database lookup

The fas antigen is involved in peripheral but not thymic deletion of T lymphocytes in T cell receptor transgenic mice

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

The role of APO-1-mediated apoptosis in the immune system

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PubMed

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https://pubmed.ncbi.nlm.nih.gov/9014814

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Expression of the Fas ligand in cells of T cell lineage.

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Expression of the functional soluble form of human fas ligand in activated lymphocytes.

1 reference

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

A soluble form of TRAP (CD40 ligand) is rapidly released after T cell activation

1 reference

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Regulation of cell surface APO-1/Fas (CD95) ligand expression by metalloproteases

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Interaction of peptides derived from the Fas ligand with the Fyn-SH3 domain

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Activation-induced death of mature T cells in the regulation of immune responses

1 reference

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Fas Ligand-Induced Apoptosis as a Mechanism of Immune Privilege

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PubMed

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https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Metalloproteinase-mediated release of human Fas ligand

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PubMed

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https://pubmed.ncbi.nlm.nih.gov/9014814

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Fas and FasL in the homeostatic regulation of immune responses

1 reference

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Fas ligand in human serum

1 reference

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Feasibility of drug screening with panels of human tumor cell lines using a microculture tetrazolium assay

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PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/9014814

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

DOI

10.1046/J.1365-2567.1996.D01-782.X

1 reference

stated in

Europe PubMed Central

PubMed publication ID

9014814

retrieved

27 September 2017

PMC publication ID

1456570

0 references

PubMed publication ID

9014814

1 reference

stated in

Europe PubMed Central

PubMed publication ID

9014814

retrieved

27 September 2017

ResearchGate publication ID

14193767

0 references

 

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