Resistance to nitric oxide-mediated apoptosis in HL-60 variant cells is associated with increased activities of Cu,Zn-superoxide dismutase and catalase (Q40979238)

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scientific article published on February 1999

Language	Label	Description	Also known as
English	Resistance to nitric oxide-mediated apoptosis in HL-60 variant cells is associated with increased activities of Cu,Zn-superoxide dismutase and catalase	scientific article published on February 1999

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

25 September 2017

Resistance to nitric oxide-mediated apoptosis in HL-60 variant cells is associated with increased activities of Cu,Zn-superoxide dismutase and catalase (English)

1 reference

Europe PubMed Central

PubMed publication ID

25 September 2017

author name string

Yabuki M

1

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25 September 2017

Kariya S

2

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25 September 2017

Ishisaka R

3

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25 September 2017

Yasuda T

4

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Europe PubMed Central

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25 September 2017

Yoshioka T

5

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Europe PubMed Central

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25 September 2017

Horton AA

6

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Europe PubMed Central

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25 September 2017

Utsumi K

7

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Europe PubMed Central

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25 September 2017

publication date

1 February 1999

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Europe PubMed Central

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25 September 2017

Free Radical Biology and Medicine

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25 September 2017

26

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25 September 2017

3-4

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25 September 2017

325-332

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25 September 2017

Apoptosis: A Basic Biological Phenomenon with Wideranging Implications in Tissue Kinetics

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Glucocorticoid-induced thymocyte apoptosis is associated with endogenous endonuclease activation

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The biochemistry of programmed cell death

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Double identity for proteins of the Bcl-2 family

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Oxygen-dependent fragmentation of cellular DNA by nitric oxide

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Molecular Mechanisms of Apoptosis in HL-60 Cells Induced by a Nitric Oxide-Releasing Compound

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Oxygen-dependent regulation of mitochondrial energy metabolism by nitric oxide

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Reversible binding and inhibition of catalase by nitric oxide

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Inactivation of glutathione peroxidase by nitric oxide. Implication for cytotoxicity

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Apparent hydroxyl radical production by peroxynitrite: implications for endothelial injury from nitric oxide and superoxide

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Peroxynitrite-induced apoptosis in HL-60 cells

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A redox-based mechanism for the neuroprotective and neurodestructive effects of nitric oxide and related nitroso-compounds

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Determination of catalase activity by means of the Clark oxygen electrode

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The reaction of no with superoxide

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Bcl-2 protects macrophages from nitric oxide-induced apoptosis

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Bcl-2 functions in an antioxidant pathway to prevent apoptosis

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Bcl-2 inhibition of neural death: decreased generation of reactive oxygen species

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Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis

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ALS, SOD and peroxynitrite

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Nitric oxide: cytotoxicity versus cytoprotection--how, why, when, and where?

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Identifiers

10.1016/S0891-5849(98)00203-2

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Europe PubMed Central

PubMed publication ID

25 September 2017

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24 November 2022

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mapped directly with Wikidata item

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

25 September 2017

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