Expression analysis of four endoglin missense mutations suggests that haploinsufficiency is the predominant mechanism for hereditary hemorrhagic telangiectasia type 1. (Q40920042)
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English | Expression analysis of four endoglin missense mutations suggests that haploinsufficiency is the predominant mechanism for hereditary hemorrhagic telangiectasia type 1. |
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Expression analysis of four endoglin missense mutations suggests that haploinsufficiency is the predominant mechanism for hereditary hemorrhagic telangiectasia type 1. (English)
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N Pece-Barbara
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U Cymerman
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S Vera
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D A Marchuk
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M Letarte
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1 November 1999
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8
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12
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2171-2181
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