Mediation of IGF-1-induced skeletal myotube hypertrophy by PI(3)K/Akt/mTOR and PI(3)K/Akt/GSK3 pathways. (Q40767544)

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3 December 2019

title

Mediation of IGF-1-induced skeletal myotube hypertrophy by PI(3)K/Akt/mTOR and PI(3)K/Akt/GSK3 pathways (English)

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3 December 2019

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3 December 2019

Rommel C

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3 December 2019

Clarke BA

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3 December 2019

Rossman R

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3 December 2019

Nunez L

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3 December 2019

Stitt TN

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3 December 2019

Yancopoulos GD

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3 December 2019

Glass DJ

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3 December 2019

language of work or name

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1 November 2001

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3 December 2019

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3 December 2019

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1009-1013

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Overexpression of hIGF-1 exclusively in skeletal muscle increases the number of dihydropyridine receptors in adult transgenic mice

3 December 2019

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IGF-1 induces skeletal myocyte hypertrophy through calcineurin in association with GATA-2 and NF-ATc1.

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Insulin-like growth factor (IGF-I) induces myotube hypertrophy associated with an increase in anaerobic glycolysis in a clonal skeletal-muscle cell model

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Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo

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