Opposite effects of Ha-Ras and Ki-Ras on radiation-induced apoptosis via differential activation of PI3K/Akt and Rac/p38 mitogen-activated protein kinase signaling pathways (Q40601308)

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scientific article published on January 2004

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English	Opposite effects of Ha-Ras and Ki-Ras on radiation-induced apoptosis via differential activation of PI3K/Akt and Rac/p38 mitogen-activated protein kinase signaling pathways	scientific article published on January 2004

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scholarly article

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Europe PubMed Central

PubMed publication ID

21 September 2017

Opposite effects of Ha-Ras and Ki-Ras on radiation-induced apoptosis via differential activation of PI3K/Akt and Rac/p38 mitogen-activated protein kinase signaling pathways (English)

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Europe PubMed Central

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21 September 2017

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Hee Yong Chung

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Jung-A Choi

1

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21 September 2017

Moon-Taek Park

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21 September 2017

Chang-Mo Kang

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21 September 2017

Hong-Duck Um

4

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21 September 2017

Sangwoo Bae

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21 September 2017

Kee-Ho Lee

6

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21 September 2017

Tae-Hwan Kim

7

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21 September 2017

Jae-Hong Kim

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21 September 2017

Chul-Koo Cho

9

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21 September 2017

Yun-Sil Lee

10

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21 September 2017

Su-Jae Lee

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21 September 2017

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publication date

1 January 2004

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21 September 2017

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21 September 2017

23

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21 September 2017

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9-20

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21 September 2017

Studies on the Influence of the Presence of an ActivatedRasOncogene on thein VitroRadiosensitivity of Human Mammary Epithelial Cells

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Caspases induce cytochrome c release from mitochondria by activating cytosolic factors

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p38 MAP kinase mediates nitric oxide-induced apoptosis of neural progenitor cells.

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Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis

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Implication of the small GTPase Rac1 in the apoptosis induced by UV in Rat-2 fibroblasts

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Bax-induced caspase activation and apoptosis via cytochrome c release from mitochondria is inhibitable by Bcl-xL.

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Regulated targeting of BAX to mitochondria.

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Apoptosis induction by T-2 toxin: activation of caspase-9, caspase-3, and DFF-40/CAD through cytosolic release of cytochrome c in HL-60 cells

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Four human ras homologs differ in their abilities to activate Raf-1, induce transformation, and stimulate cell motility

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Differential activation of the Rac pathway by Ha-Ras and K-Ras

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Oncogenic Ki-ras but not oncogenic Ha-ras blocks integrin beta1-chain maturation in colon epithelial cells

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Ras-induced cellular events (review)

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Increasing complexity of Ras signaling.

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Enhanced sensitivity to apoptosis in Ras-transformed thyroid cells

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Roles of JNK, p38 and ERK mitogen-activated protein kinases in the growth inhibition and apoptosis induced by cadmium

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Ras proteins in the control of the cell cycle and cell differentiation

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Protection of human endothelial cells from oxidative stress: role of Ras-ERK1/2 signaling.

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Cellular survival: a play in three Akts

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Activated human N-ras oncogene enhances x-irradiation repair of mammalian cells in vitro less effectively at low dose rate. Implications for increased therapeutic ratio of low dose rate irradiation

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Stimulation of p38 mitogen-activated protein kinase is an early regulatory event for the cadmium-induced apoptosis in human promonocytic cells

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p38 MAP kinase mediates bax translocation in nitric oxide-induced apoptosis in neurons

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RAS-Mediated radiation resistance is not linked to MAP kinase activation in two bladder carcinoma cell lines.

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Some ras-transformed cells have increased radiosensitivity and decreased repair of sublethal radiation damage

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A phosphatidylinositol 3-kinase inhibitor wortmannin induces radioresistant DNA synthesis and sensitizes cells to bleomycin and ionizing radiation

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Matrix adhesion and Ras transformation both activate a phosphoinositide 3-OH kinase and protein kinase B/Akt cellular survival pathway

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Antiapoptotic signalling by the insulin-like growth factor I receptor, phosphatidylinositol 3-kinase, and Akt.

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Redox regulation of cell signalling

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Radioresistance induced by oncogenic transformation

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Increased radioresistance of EJras-transformed human osteosarcoma cells and its modulation by lovastatin, an inhibitor of p21ras isoprenylation

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Superoxides relay Ras protein's oncogenic message

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Activation of the RacGTPase inhibits apoptosis in human tumor cells

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Transforming G proteins

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The cdc2 kinase is a nuclear protein that is essential for mitosis in mammalian cells

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Opposing functions of Ki- and Ha-Ras genes in the regulation of redox signals.

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The ras oncogenes increase the intrinsic resistance of NIH 3T3 cells to ionizing radiation

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Ras isoforms vary in their ability to activate Raf-1 and phosphoinositide 3-kinase

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Requirement for phosphatidylinositol-3 kinase in the prevention of apoptosis by nerve growth factor

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Differential involvement of p38 MAP kinase pathway and Bax translocation in the mitochondria-mediated cell death in TCR- and dexamethasone-stimulated thymocytes.

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10.1038/SJ.ONC.1206982

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Europe PubMed Central

PubMed publication ID

21 September 2017

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PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

21 September 2017

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