Endoplasmic reticulum stress stimulates the expression of cyclooxygenase-2 through activation of NF-kappaB and pp38 mitogen-activated protein kinase (Q40524016)

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scientific article published on 19 August 2004

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English	Endoplasmic reticulum stress stimulates the expression of cyclooxygenase-2 through activation of NF-kappaB and pp38 mitogen-activated protein kinase	scientific article published on 19 August 2004

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scholarly article

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Europe PubMed Central

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21 September 2017

Endoplasmic reticulum stress stimulates the expression of cyclooxygenase-2 through activation of NF-kappaB and pp38 mitogen-activated protein kinase (English)

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21 September 2017

endoplasmic reticulum

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Jui-Hsiang Hung

1

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21 September 2017

2

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Huan-Yao Lei

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21 September 2017

Hui-Ching Wang

4

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21 September 2017

Wan-Chi Lin

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21 September 2017

Wen-Tsan Chang

6

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21 September 2017

Wenya Huang

7

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21 September 2017

Wen-Chang Chang

8

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21 September 2017

Yung-Sheng Chang

9

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21 September 2017

Ching-Chow Chen

10

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21 September 2017

Ming-Derg Lai

11

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21 September 2017

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publication date

19 August 2004

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21 September 2017

Journal of Biological Chemistry

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21 September 2017

279

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21 September 2017

45

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21 September 2017

46384-46392

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21 September 2017

Intracellular signaling from the endoplasmic reticulum to the nucleus: the unfolded protein response in yeast and mammals

1 reference

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The endoplasmic reticulum: integration of protein folding, quality control, signaling and degradation.

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Endoplasmic reticulum dysfunction--a common denominator for cell injury in acute and degenerative diseases of the brain?

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Cellular defenses against unfolded proteins: a cell biologist thinks about neurodegenerative diseases

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When translation meets metabolism: multiple links to diabetes

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Pre-S mutant surface antigens in chronic hepatitis B virus infection induce oxidative stress and DNA damage

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Different types of ground glass hepatocytes in chronic hepatitis B virus infection contain specific pre-S mutants that may induce endoplasmic reticulum stress

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Hepatitis C virus subgenomic replicons induce endoplasmic reticulum stress activating an intracellular signaling pathway

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Endoplasmic reticulum (ER) stress: hepatitis C virus induces an ER-nucleus signal transduction pathway and activates NF-kappaB and STAT-3.

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Japanese encephalitis virus infection initiates endoplasmic reticulum stress and an unfolded protein response

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Mammalian transcription factor ATF6 is synthesized as a transmembrane protein and activated by proteolysis in response to endoplasmic reticulum stress

1 reference

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21 January 2018

ATF6 activated by proteolysis binds in the presence of NF-Y (CBF) directly to the cis-acting element responsible for the mammalian unfolded protein response

1 reference

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ATF6 as a transcription activator of the endoplasmic reticulum stress element: thapsigargin stress-induced changes and synergistic interactions with NF-Y and YY1

1 reference

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XBP1 mRNA is induced by ATF6 and spliced by IRE1 in response to ER stress to produce a highly active transcription factor

1 reference

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21 January 2018

Complementary signaling pathways regulate the unfolded protein response and are required for C. elegans development

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IRE1 couples endoplasmic reticulum load to secretory capacity by processing the XBP-1 mRNA

1 reference

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IRE1-mediated unconventional mRNA splicing and S2P-mediated ATF6 cleavage merge to regulate XBP1 in signaling the unfolded protein response

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21 January 2018

Protein translation and folding are coupled by an endoplasmic-reticulum-resident kinase

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Identification and characterization of pancreatic eukaryotic initiation factor 2 alpha-subunit kinase, PEK, involved in translational control

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The ER-overload response: activation of NF-kappa B.

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Activation of transcription factor NF-kappaB by the adenovirus E3/19K protein requires its ER retention

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Endoplasmic reticulum stress causes thyroglobulin retention in this organelle and triggers activation of nuclear factor-kappa B via tumor necrosis factor receptor-associated factor 2.

1 reference

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21 January 2018

Activation signal of nuclear factor-kappa B in response to endoplasmic reticulum stress is transduced via IRE1 and tumor necrosis factor receptor-associated factor 2

1 reference

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21 January 2018

Phosphorylation of the alpha subunit of eukaryotic initiation factor 2 is required for activation of NF-kappaB in response to diverse cellular stresses

1 reference

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21 January 2018

Many actions of cyclooxygenase-2 in cellular dynamics and in cancer

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21 January 2018

Correlation of cyclooxygenase-2 expression with molecular markers, pathological features and clinical outcome of transitional cell carcinoma of the bladder

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The hepatitis B virus X protein promotes tumor cell invasion by inducing membrane-type matrix metalloproteinase-1 and cyclooxygenase-2 expression

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21 January 2018

Transgenic cyclooxygenase-2 overexpression sensitizes mouse skin for carcinogenesis

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21 January 2018

The induction of cyclooxygenase-2 mRNA in macrophages is biphasic and requires both CCAAT enhancer-binding protein beta (C/EBP beta ) and C/EBP delta transcription factors

1 reference

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21 January 2018

Redundancy in the signaling pathways and promoter elements regulating cyclooxygenase-2 gene expression in endotoxin-treated macrophage/monocytic cells

1 reference

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21 January 2018

Ceramide-induced and age-associated increase in macrophage COX-2 expression is mediated through up-regulation of NF-kappa B activity

1 reference

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21 January 2018

Myeloid differentiation factor 88-dependent transcriptional regulation of cyclooxygenase-2 expression by CpG DNA: role of NF-kappaB and p38.

1 reference

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21 January 2018

Upregulation of cyclooxygenase-2 is accompanied by increased expression of nuclear factor-kappa B and I kappa B kinase-alpha in human colorectal cancer epithelial cells

1 reference

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21 January 2018

Leptomycin B, an inhibitor of the nuclear export receptor CRM1, inhibits COX-2 expression

1 reference

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21 January 2018

Prevalence and significance of hepatitis B virus (HBV) pre-S mutants in serum and liver at different replicative stages of chronic HBV infection

1 reference

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21 January 2018

Herp is dually regulated by both the endoplasmic reticulum stress-specific branch of the unfolded protein response and a branch that is shared with other cellular stress pathways

1 reference

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Nck-dependent activation of extracellular signal-regulated kinase-1 and regulation of cell survival during endoplasmic reticulum stress

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Suppression of RelA/p65 nuclear translocation independent of IkappaB-alpha degradation by cyclooxygenase-2 inhibitor in gastric cancer

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Synergistic activation of NF-kappaB by nontypeable Haemophilus influenzae and tumor necrosis factor alpha

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Activation of discoidin domain receptor 1 isoform b with collagen up-regulates chemokine production in human macrophages: role of p38 mitogen-activated protein kinase and NF-kappa B.

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Prostaglandin E(2) regulates the level and stability of cyclooxygenase-2 mRNA through activation of p38 mitogen-activated protein kinase in interleukin-1 beta-treated human synovial fibroblasts.

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p38-Dependent marking of inflammatory genes for increased NF-kappa B recruitment

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Endoplasmic reticulum stress induces p53 cytoplasmic localization and prevents p53-dependent apoptosis by a pathway involving glycogen synthase kinase-3beta

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Expression of the pro-angiogenic factors vascular endothelial growth factor and interleukin-8/CXCL8 by human breast carcinomas is responsive to nutrient deprivation and endoplasmic reticulum stress

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Glycogen Synthase Kinase-3β (GSK3β) Binds to and Promotes the Actions of p53

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Betulinic acid suppresses carcinogen-induced NF-kappa B activation through inhibition of I kappa B alpha kinase and p65 phosphorylation: abrogation of cyclooxygenase-2 and matrix metalloprotease-9.

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Exploiting hepatitis C virus activation of NFκB to deliver HCV-responsive expression of interferons α and γ

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Colorectal cancer prevention and treatment by inhibition of cyclooxygenase-2.

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Proapoptotic and antiproliferative potential of selective cyclooxygenase-2 inhibitors in human liver tumor cells

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NS-398, a selective cyclooxygenase 2 inhibitor, inhibited cell growth and induced cell cycle arrest in human hepatocellular carcinoma cell lines

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Hepatocellular carcinoma: is there a potential for chemoprevention using cyclooxygenase-2 inhibitors?

1 reference

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JTE-522, a cyclooxygenase-2 inhibitor, is an effective chemopreventive agent against rat experimental liver fibrosis1.

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21 January 2018

Identifiers

10.1074/JBC.M403568200

1 reference

Europe PubMed Central

PubMed publication ID

21 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

21 September 2017

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