Bisphosphonates and estrogens inhibit osteocyte apoptosis via distinct molecular mechanisms downstream of extracellular signal-regulated kinase activation. (Q40482310)

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English	Bisphosphonates and estrogens inhibit osteocyte apoptosis via distinct molecular mechanisms downstream of extracellular signal-regulated kinase activation.	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

20 September 2017

Bisphosphonates and estrogens inhibit osteocyte apoptosis via distinct molecular mechanisms downstream of extracellular signal-regulated kinase activation. (English)

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Europe PubMed Central

PubMed publication ID

20 September 2017

apoptotic process

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Teresita M. Bellido

5

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Teresita Bellido

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Europe PubMed Central

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20 September 2017

author name string

Lilian I Plotkin

1

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20 September 2017

J Ignacio Aguirre

2

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20 September 2017

Stavroula Kousteni

3

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20 September 2017

Stavros C Manolagas

4

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20 September 2017

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publication date

6 December 2004

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20 September 2017

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9

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Journal of Biological Chemistry

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20 September 2017

280

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20 September 2017

8

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20 September 2017

7317-7325

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20 September 2017

Prevention of osteocyte and osteoblast apoptosis by bisphosphonates and calcitonin

1 reference

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Transduction of cell survival signals by connexin-43 hemichannels

1 reference

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Tyrosine Phosphorylation of Connexin 43 by v-Src Is Mediated by SH2 and SH3 Domain Interactions

1 reference

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Kinase-mediated regulation of common transcription factors accounts for the bone-protective effects of sex steroids

1 reference

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Growth factors inactivate the cell death promoter BAD by phosphorylation of its BH3 domain on Ser155.

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A Dominant-Negative Inhibitor of CREB Reveals that It Is a General Mediator of Stimulus-Dependent Transcription of c-fos

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A Cbfa1-dependent genetic pathway controls bone formation beyond embryonic development

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Dominant negative stat3 mutant inhibits interleukin-6-induced Jak-STAT signal transduction

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Akt Phosphorylation of BAD Couples Survival Signals to the Cell-Intrinsic Death Machinery

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C/EBPbeta phosphorylation by RSK creates a functional XEXD caspase inhibitory box critical for cell survival

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Estrogen receptor-mediated activation of the serum response element in MCF-7 cells through MAPK-dependent phosphorylation of Elk-1.

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Stimulation of Elk1 transcriptional activity by mitogen-activated protein kinases is negatively regulated by protein phosphatase 2B (calcineurin).

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Dissection of the molecular basis of pp60(v-src) induced gating of connexin 43 gap junction channels

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Establishment of an osteocyte-like cell line, MLO-Y4.

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S6 kinase p90rsk in granulocyte-macrophage colony-stimulating factor-stimulated proliferative and mature hematopoietic cells

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Cell survival promoted by the Ras-MAPK signaling pathway by transcription-dependent and -independent mechanisms

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Regulation of bad phosphorylation and association with Bcl-x(L) by the MAPK/Erk kinase

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ERK activation induces phosphorylation of Elk-1 at multiple S/T-P motifs to high stoichiometry

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Transcriptional regulation by MAP kinases

1 reference

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Glutamate induces phosphorylation of Elk-1 and CREB, along with c-fos activation, via an extracellular signal-regulated kinase-dependent pathway in brain slices

1 reference

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21 January 2018

MAPK pathways activate and phosphorylate the osteoblast-specific transcription factor, Cbfa1.

1 reference

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Biphasic regulation of extracellular-signal-regulated protein kinase by leptin in macrophages: role in regulating STAT3 Ser727 phosphorylation and DNA binding

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Trichostatin and leptomycin. Inhibition of histone deacetylation and signal-dependent nuclear export

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Role and regulation of 90 kDa ribosomal S6 kinase (RSK) in signal transduction.

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Insulin-stimulated MAP-2 kinase phosphorylates and activates ribosomal protein S6 kinase II.

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IEX-1: a new ERK substrate involved in both ERK survival activity and ERK activation

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Molecular interpretation of ERK signal duration by immediate early gene products

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Rsk1 mediates a MEK-MAP kinase cell survival signal

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Cytosolic retention of phosphorylated extracellular signal-regulated kinase and a Rho-associated kinase-mediated signal impair expression of p21(Cip1/Waf1) in phorbol 12-myristate-13- acetate-induced apoptotic cells

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Subcellular localization determines the protective effects of activated ERK2 against distinct apoptogenic stimuli in myeloid leukemia cells.

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Regulation of Ras-MAPK pathway mitogenic activity by restricting nuclear entry of activated MAPK in endoderm differentiation of embryonic carcinoma and stem cells

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RhoA influences the nuclear localization of extracellular signal-regulated kinases to modulate p21Waf/Cip1 expression

1 reference

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The stability of the G protein-coupled receptor-beta-arrestin interaction determines the mechanism and functional consequence of ERK activation

1 reference

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Sef is a spatial regulator for Ras/MAP kinase signaling

1 reference

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21 January 2018

Glucocorticoids act directly on osteoblasts and osteocytes to induce their apoptosis and reduce bone formation and strength

1 reference

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Increased bone formation by prevention of osteoblast apoptosis with parathyroid hormone

1 reference

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Proteasomal degradation of Runx2 shortens parathyroid hormone-induced anti-apoptotic signaling in osteoblasts. A putative explanation for why intermittent administration is needed for bone anabolism

1 reference

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Reversal of bone loss in mice by nongenotropic signaling of sex steroids

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Fas/CD95 is associated with glucocorticoid-induced osteocyte apoptosis

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Effect of combined risedronate and hormone replacement therapies on bone mineral density in postmenopausal women

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21 January 2018

Identifiers

10.1074/JBC.M412817200

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

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