Genome-wide response of the human Hep3B hepatoma cell to proinflammatory cytokines, from transcription to translation (Q40370668)

19 September 2017

title

Genome-wide response of the human Hep3B hepatoma cell to proinflammatory cytokines, from transcription to translation (English)

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19 September 2017

main subject

hepatocellular carcinoma

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Cédric Coulouarn

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19 September 2017

Grégory Lefebvre

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19 September 2017

Romain Daveau

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19 September 2017

Franck Letellier

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19 September 2017

Martine Hiron

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19 September 2017

Laurent Drouot

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19 September 2017

Maryvonne Daveau

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19 September 2017

Jean-Philippe Salier

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19 September 2017

language of work or name

English

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publication date

1 October 2005

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19 September 2017

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19 September 2017

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19 September 2017

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19 September 2017

page(s)

946-955

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Molecular signatures of sepsis: multiorgan gene expression profiles of systemic inflammation

19 September 2017

cites work

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Kinetic modeling and mathematical analysis indicate that acute phase gene expression in Hep 3B cells is regulated by both transcriptional and posttranscriptional mechanisms

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Innate and acquired immunity intersect in a global view of the acute-phase response

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Temporal control of NF-kappaB activation by ERK differentially regulates interleukin-1beta-induced gene expression.

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Transcription factors Stat3 and Stat5b are present in rat liver nuclei late in an acute phase response and bind interleukin-6 response elements

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The cell death regulator GRIM-19 is an inhibitor of signal transducer and activator of transcription 3

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Comparing protein abundance and mRNA expression levels on a genomic scale

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Control of pancreas and liver gene expression by HNF transcription factors

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Genomic run-on evaluates transcription rates for all yeast genes and identifies gene regulatory mechanisms

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Global mRNA stabilization preferentially linked to translational repression during the endoplasmic reticulum stress response

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Ubiquitin-dependent mechanism regulates rapid turnover of AU-rich cytokine mRNAs

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Distinct domains of AU-rich elements exert different functions in mRNA destabilization and stabilization by p38 mitogen-activated protein kinase or HuR.

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Liver gene expression associated with diet and lesion development in atherosclerosis-prone mice: induction of components of alternative complement pathway.

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Identifiers

DOI

10.1002/HEP.20848

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19 September 2017

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