MLL chimeric protein activation renders cells vulnerable to chromosomal damage: an explanation for the very short latency of infant leukemia (Q40281320)

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scientific article published on August 2006

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English	MLL chimeric protein activation renders cells vulnerable to chromosomal damage: an explanation for the very short latency of infant leukemia	scientific article published on August 2006

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

MLL chimeric protein activation renders cells vulnerable to chromosomal damage: an explanation for the very short latency of infant leukemia (English)

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Europe PubMed Central

PubMed publication ID

19 September 2017

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object named as

Mel Greaves

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19 September 2017

author name string

Mariko Eguchi

1

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Europe PubMed Central

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19 September 2017

Minenori Eguchi-Ishimae

2

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19 September 2017

Deborah Knight

3

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19 September 2017

Lyndal Kearney

4

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Europe PubMed Central

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19 September 2017

Robert Slany

5

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Europe PubMed Central

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19 September 2017

publication date

1 August 2006

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Europe PubMed Central

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19 September 2017

Genes, Chromosomes and Cancer

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Europe PubMed Central

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19 September 2017

45

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19 September 2017

8

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19 September 2017

754-760

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Europe PubMed Central

PubMed publication ID

19 September 2017

Leukemic HRX fusion proteins inhibit GADD34-induced apoptosis and associate with the GADD34 and hSNF5/INI1 proteins

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

21 January 2018

Secondary leukemias induced by topoisomerase-targeted drugs

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

21 January 2018

Molecular mechanisms of leukemogenesis mediated by MLL fusion proteins

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https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

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inferred from DOI database lookup

Modulation of cell cycle by graded expression of MLL-AF4 fusion oncoprotein

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

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An Mll-AF9 fusion gene made by homologous recombination causes acute leukemia in chimeric mice: a method to create fusion oncogenes

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

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Molecular pathogenesis of MLL-associated leukemias

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

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In utero rearrangements in the trithorax-related oncogene in infant leukaemias.

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

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Backtracking leukemia to birth: Identification of clonotypic gene fusion sequences in neonatal blood spots

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

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Leukemia in twins: lessons in natural history

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https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

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Absence of p53 permits propagation of mutant cells following genotoxic damage

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

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An atlas of chromosomes in hematological malignancies. Example: 11q23 and MLL partners

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

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Prenatal and postnatal myeloid cells demonstrate stepwise progression in the pathogenesis of MLL fusion gene leukemia

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

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p53-deficient mice are extremely susceptible to radiation-induced tumorigenesis

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

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Immortalization and leukemic transformation of a myelomonocytic precursor by retrovirally transduced HRX-ENL

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

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Therapy-related acute myeloid leukemia-like MLL rearrangements are induced by etoposide in primary human CD34+ cells and remain stable after clonal expansion

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

inferred from DOI database lookup

Protracted postnatal natural histories in childhood leukemia

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

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The translocations, t(11;19)(q23;p13.1) and t(11;19)(q23;p13.3): a cytogenetic and clinical profile of 53 patients. European 11q23 Workshop participants

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

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Expression of leukemic MLL fusion proteins in Drosophila affects cell cycle control and chromosome morphology.

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

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Maternal exposure to potential inhibitors of DNA topoisomerase II and infant leukemia (United States): a report from the Children's Cancer Group.

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

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International workshop on the relationship of prior therapy to balanced chromosome aberrations in therapy-related myelodysplastic syndromes and acute leukemia: overview report

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

inferred from DOI database lookup

When epigenetics kills: MLL fusion proteins in leukemia

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

inferred from DOI database lookup

Dietary bioflavonoids induce cleavage in the MLL gene and may contribute to infant leukemia

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

inferred from DOI database lookup

FLT3 mutations in the activation loop of tyrosine kinase domain are frequently found in infant ALL with MLL rearrangements and pediatric ALL with hyperdiploidy

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

inferred from DOI database lookup

Aborting the birth of cancer

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

inferred from DOI database lookup

Conditional MLL-CBP targets GMP and models therapy-related myeloproliferative disease.

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

inferred from DOI database lookup

Decoding the links between mitosis, cancer, and chemotherapy: The mitotic checkpoint, adaptation, and cell death.

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

inferred from DOI database lookup

Multiple mixed lineage leukemia (MLL) fusion proteins suppress p53-mediated response to DNA damage

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

inferred from DOI database lookup

The MLL fusion gene, MLL-AF4, regulates cyclin-dependent kinase inhibitor CDKN1B (p27kip1) expression

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

inferred from DOI database lookup

Hoxa9 and Meis1 are key targets for MLL-ENL-mediated cellular immortalization

1 reference

https://api.crossref.org/works/10.1002%2FGCC.20338

7 January 2021

based on heuristic

inferred from DOI database lookup

Identifiers

10.1002/GCC.20338

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

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