Expression of alpha V integrin is modulated by Epstein-Barr virus nuclear antigen 3C and the metastasis suppressor Nm23-H1 through interaction with the GATA-1 and Sp1 transcription factors (Q40288802)

From Wikidata

Jump to navigation Jump to search

scientific article published on 2 May 2006

Language	Label	Description	Also known as
English	Expression of alpha V integrin is modulated by Epstein-Barr virus nuclear antigen 3C and the metastasis suppressor Nm23-H1 through interaction with the GATA-1 and Sp1 transcription factors	scientific article published on 2 May 2006

Statements

scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

Expression of alpha V integrin is modulated by Epstein-Barr virus nuclear antigen 3C and the metastasis suppressor Nm23-H1 through interaction with the GATA-1 and Sp1 transcription factors (English)

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

Epstein–Barr virus

0 references

2

1 reference

Europe PubMed Central

PubMed publication ID

9 November 2018

Erle S Robertson

4

object named as

Erle S Robertson

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

author name string

Tathagata Choudhuri

1

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

Ke Lan

3

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

language of work or name

0 references

publication date

2 May 2006

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

351

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

1

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

58-72

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

Murine erythroleukemia cells contain two distinct GATA-binding proteins that have different patterns of expression during cellular differentiation

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Localization of matrix metalloproteinase MMP-2 to the surface of invasive cells by interaction with integrin alpha v beta 3.

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Insulin-like growth factor receptor cooperates with integrin alpha v beta 5 to promote tumor cell dissemination in vivo

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

The vitronectin receptor alpha v beta 3 binds fibronectin and acts in concert with alpha 5 beta 1 in promoting cellular attachment and spreading on fibronectin

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Integrins in thrombosis, wound healing and cancer

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Epstein-Barr virus nuclear protein 2 is a key determinant of lymphocyte transformation

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

The role of alpha(v)beta(3) in prostate cancer progression

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Selective Sp1 and Sp3 binding is crucial for activity of the integrin alphaV promoter in cultured endothelial cells

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Osteopontin: a protein with diverse functions.

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Requirement of the NPXY motif in the integrin beta 3 subunit cytoplasmic tail for melanoma cell migration in vitro and in vivo.

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Cell growth and matrix invasion of EBV-immortalized human B lymphocytes is regulated by expression of alpha(v) integrins

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Integrins: versatility, modulation, and signaling in cell adhesion

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Epstein-Barr virus latent membrane protein 1 is essential for B-lymphocyte growth transformation

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

The latency-associated nuclear antigen of Kaposi's sarcoma-associated herpesvirus transactivates the telomerase reverse transcriptase promoter

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Regulation of matrix metalloproteinase 9 expression by Epstein-Barr virus nuclear antigen 3C and the suppressor of metastasis Nm23-H1.

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

The human Nm23/nucleoside diphosphate kinases

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Kaposi's sarcoma-associated herpesvirus-encoded latency-associated nuclear antigen inhibits lytic replication by targeting Rta: a potential mechanism for virus-mediated control of latency

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Reduced tumor incidence, metastatic potential, and cytokine responsiveness of nm23-transfected melanoma cells

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Epstein-Barr virus nuclear antigen 3C putative repression domain mediates coactivation of the LMP1 promoter with EBNA-2.

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

NM23-H1 and NM23-H2 repress transcriptional activities of nuclease-hypersensitive elements in the platelet-derived growth factor-A promoter

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Cellular adhesion pathways and metastatic potential of human melanoma

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Coordinated expression of the vitronectin receptor and the urokinase-type plasminogen activator receptor in metastatic melanoma cells

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Focal adhesion kinase: the first ten years

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

A 125/115-kDa cell surface receptor specific for vitronectin interacts with the arginine-glycine-aspartic acid adhesion sequence derived from fibronectin

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Integrin signaling and matrix assembly

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Matrix metalloproteinases in tumor invasion and metastasis

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

The metastatic suppressor Nm23-H1 interacts with EBNA3C at sequences located between the glutamine- and proline-rich domains and can cooperate in activation of transcription

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Epstein-Barr virus nuclear protein EBNA-3C interacts with the human metastatic suppressor Nm23-H1: a molecular link to cancer metastasis

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Epstein-Barr virus nuclear antigen 3C and prothymosin alpha interact with the p300 transcriptional coactivator at the CH1 and CH3/HAT domains and cooperate in regulation of transcription and histone acetylation.

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

The Epstein Barr nuclear antigen EBNA3C regulates transcription, cell transformation and cell migration

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Epstein-Barr virus and oncogenesis: from latent genes to tumours

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

The alpha v beta 1 integrin functions as a fibronectin receptor but does not support fibronectin matrix assembly and cell migration on fibronectin

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Epstein-barr virus nuclear antigen 3C activates the latent membrane protein 1 promoter in the presence of Epstein-Barr virus nuclear antigen 2 through sequences encompassing an spi-1/Spi-B binding site

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

Epstein-Barr virus EBNA3C represses Cp, the major promoter for EBNA expression, but has no effect on the promoter of the cell gene CD21.

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

based on heuristic

inferred from DOI database lookup

Epstein-Barr virus nuclear protein 3C modulates transcription through interaction with the sequence-specific DNA-binding protein J kappa

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

based on heuristic

inferred from DOI database lookup

A conserved domain of the Epstein-Barr virus nuclear antigens 3A and 3C binds to a discrete domain of Jkappa

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2006.03.031

7 January 2021

based on heuristic

inferred from DOI database lookup

Identifiers

10.1016/J.VIROL.2006.03.031

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

ResearchGate publication ID

0 references

Sitelinks

Wikipedia(0 entries)

Wikibooks(0 entries)

Wikinews(0 entries)

Wikiquote(0 entries)

Wikisource(0 entries)

Wikiversity(0 entries)

Wikivoyage(0 entries)

Wiktionary(0 entries)

Multilingual sites(0 entries)

Retrieved from "https://www.wikidata.org/w/index.php?title=Q40288802&oldid=1893493134"