The requirement for SNF5/INI1 in adipocyte differentiation highlights new features of malignant rhabdoid tumors (Q40070769)

17 September 2017

title

The requirement for SNF5/INI1 in adipocyte differentiation highlights new features of malignant rhabdoid tumors (English)

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17 September 2017

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J Caramel

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O Delattre

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17 September 2017

S Medjkane

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17 September 2017

F Quignon

series ordinal

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17 September 2017

language of work or name

English

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publication date

8 October 2007

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17 September 2017

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17 September 2017

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17 September 2017

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17 September 2017

page(s)

2035-2044

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The tumour suppressor hSNF5/INI1 controls the differentiation potential of malignant rhabdoid cells

17 September 2017

cites work

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The SWI/SNF chromatin-remodeling complex subunit SNF5 is essential for hepatocyte differentiation

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A CCAAT/enhancer binding protein beta isoform, liver-enriched inhibitory protein, regulates commitment of osteoblasts and adipocytes

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Binding and stimulation of HIV-1 integrase by a human homolog of yeast transcription factor SNF5

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The chromatin remodeler Mi-2beta is required for establishment of the basal epidermis and normal differentiation of its progeny

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The murine SNF5/INI1 chromatin remodeling factor is essential for embryonic development and tumor suppression

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Increased DNA damage sensitivity and apoptosis in cells lacking the Snf5/Ini1 subunit of the SWI/SNF chromatin remodeling complex

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A C/EBP beta isoform recruits the SWI/SNF complex to activate myeloid genes

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Cell shape, cytoskeletal tension, and RhoA regulate stem cell lineage commitment

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The tumor suppressor hSNF5/INI1 modulates cell growth and actin cytoskeleton organization

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A human protein with homology to Saccharomyces cerevisiae SNF5 interacts with the potential helicase hbrm

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Cooperation between C/EBPalpha TBP/TFIIB and SWI/SNF recruiting domains is required for adipocyte differentiation

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Constitutional mutations of the hSNF5/INI1 gene predispose to a variety of cancers

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Purification and characterization of mSin3A-containing Brg1 and hBrm chromatin remodeling complexes

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Neuroepithelial cells supply an initial transient wave of MSC differentiation.

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Truncating mutations of hSNF5/INI1 in aggressive paediatric cancer

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Cancer-associated mutations in chromatin remodeler hSNF5 promote chromosomal instability by compromising the mitotic checkpoint.

21 January 2018

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SWI/SNF chromatin remodeling complex is obligatory for BMP2-induced, Runx2-dependent skeletal gene expression that controls osteoblast differentiation

21 January 2018

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Cell cycle arrest and repression of cyclin D1 transcription by INI1/hSNF5.

21 January 2018

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Tumor-specific cooperation of retinoblastoma protein family and Snf5 inactivation

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inferred from DOI database lookup

7 January 2021

based on heuristic

Highly penetrant, rapid tumorigenesis through conditional inversion of the tumor suppressor gene Snf5

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1210847

inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1038/SJ.ONC.1210847

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Dimensions Publication ID

17 September 2017

1052819372

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1 reference