Reduction of pp32 expression in poorly differentiated pancreatic ductal adenocarcinomas and intraductal papillary mucinous neoplasms with moderate dysplasia (Q40074127)

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English	Reduction of pp32 expression in poorly differentiated pancreatic ductal adenocarcinomas and intraductal papillary mucinous neoplasms with moderate dysplasia	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

17 September 2017

Reduction of pp32 expression in poorly differentiated pancreatic ductal adenocarcinomas and intraductal papillary mucinous neoplasms with moderate dysplasia (English)

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Europe PubMed Central

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17 September 2017

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Charles J Yeo

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17 September 2017

author name string

Jonathan R Brody

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17 September 2017

Agnes Witkiewicz

2

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17 September 2017

Timothy K Williams

3

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17 September 2017

Shrihari S Kadkol

4

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17 September 2017

Joseph Cozzitorto

5

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17 September 2017

Brandice Durkan

6

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17 September 2017

Gary R Pasternack

7

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17 September 2017

publication date

28 September 2007

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17 September 2017

Modern Pathology

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17 September 2017

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17 September 2017

1238-1244

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17 September 2017

Expression of pp32 gene family members in breast cancer

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https://api.crossref.org/works/10.1038%2FMODPATHOL.3800974

21 January 2018

Identification of sequences required for inhibition of oncogene-mediated transformation by pp32.

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https://api.crossref.org/works/10.1038%2FMODPATHOL.3800974

21 January 2018

Structure of pp32, an acidic nuclear protein which inhibits oncogene-induced formation of transformed foci

1 reference

https://api.crossref.org/works/10.1038%2FMODPATHOL.3800974

21 January 2018

Tumor suppression and potentiation by manipulation of pp32 expression

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https://api.crossref.org/works/10.1038%2FMODPATHOL.3800974

21 January 2018

Phosphorylated retinoblastoma protein complexes with pp32 and inhibits pp32-mediated apoptosis

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https://api.crossref.org/works/10.1038%2FMODPATHOL.3800974

21 January 2018

Protein ligands to HuR modulate its interaction with target mRNAs in vivo

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https://api.crossref.org/works/10.1038%2FMODPATHOL.3800974

21 January 2018

Tumor suppressor pp32 represses cell growth through inhibition of transcription by blocking acetylation and phosphorylation of histone H3 and initiating its proapoptotic activity

1 reference

https://api.crossref.org/works/10.1038%2FMODPATHOL.3800974

21 January 2018

Regulation of histone acetylation and transcription by nuclear protein pp32, a subunit of the INHAT complex.

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https://api.crossref.org/works/10.1038%2FMODPATHOL.3800974

21 January 2018

Modulation of oncogenic potential by alternative gene use in human prostate cancer

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https://api.crossref.org/works/10.1038%2FMODPATHOL.3800974

21 January 2018

pp32 reduction induces differentiation of TSU-Pr1 cells

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https://api.crossref.org/works/10.1038%2FMODPATHOL.3800974

21 January 2018

Molecular genetics of pancreatic intraepithelial neoplasia

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https://api.crossref.org/works/10.1038%2FMODPATHOL.3800974

21 January 2018

Prevalence of activating K-ras mutations in the evolutionary stages of neoplasia in intraductal papillary mucinous tumors of the pancreas.

1 reference

https://api.crossref.org/works/10.1038%2FMODPATHOL.3800974

21 January 2018

Novel nuclear phosphoprotein pp32 is highly expressed in intermediate- and high-grade prostate cancer

1 reference

https://api.crossref.org/works/10.1038%2FMODPATHOL.3800974

7 January 2021

based on heuristic

inferred from DOI database lookup

HuR: post-transcriptional paths to malignancy.

1 reference

https://api.crossref.org/works/10.1038%2FMODPATHOL.3800974

7 January 2021

based on heuristic

inferred from DOI database lookup

1423 pancreaticoduodenectomies for pancreatic cancer: A single-institution experience

1 reference

https://api.crossref.org/works/10.1038%2FMODPATHOL.3800974

7 January 2021

based on heuristic

inferred from DOI database lookup

Identifiers

10.1038/MODPATHOL.3800974

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Europe PubMed Central

PubMed publication ID

17 September 2017

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PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

17 September 2017

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