Wnt/beta-catenin signaling stimulates matrix catabolic genes and activity in articular chondrocytes: its possible role in joint degeneration (Q40019581)

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scientific article published on 28 January 2008

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English	Wnt/beta-catenin signaling stimulates matrix catabolic genes and activity in articular chondrocytes: its possible role in joint degeneration	scientific article published on 28 January 2008

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

Wnt/beta-catenin signaling stimulates matrix catabolic genes and activity in articular chondrocytes: its possible role in joint degeneration (English)

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

author name string

Takahito Yuasa

1

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

Tomohiro Otani

2

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

Tatsuya Koike

3

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

Masahiro Iwamoto

4

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

Motomi Enomoto-Iwamoto

5

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

publication date

28 January 2008

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

Laboratory Investigation

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Europe PubMed Central

PubMed publication ID

16 September 2017

88

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

3

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

264-274

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

1 reference

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Wnt signalling: pathway or network?

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Wnt signaling: a common theme in animal development

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WNTs modulate cell fate and behavior during vertebrate development

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A new member of the frizzled family from Drosophila functions as a Wingless receptor

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Opposing roles of WNT-5A and WNT-11 in interleukin-1beta regulation of type II collagen expression in articular chondrocytes

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Alteration of matrix glycosaminoglycans diminishes articular chondrocytes' response to a canonical Wnt signal

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Impaired endochondral ossification and angiogenesis in mice deficient in membrane-type matrix metalloproteinase I

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Critical roles for collagenase-3 (Mmp13) in development of growth plate cartilage and in endochondral ossification

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Bone morphogenetic protein signaling is required for maintenance of differentiated phenotype, control of proliferation, and hypertrophy in chondrocytes

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Wnt/beta-catenin/Tcf signaling induces the transcription of Axin2, a negative regulator of the signaling pathway

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Retinoic acid induces rapid mineralization and expression of mineralization-related genes in chondrocytes

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Effects of hyaluronic acid on the release of proteoglycan from the cell matrix in rabbit chondrocyte cultures in the presence and absence of cytokines

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Selective activation of the mitogen-activated protein kinase subgroups c-Jun NH2 terminal kinase and p38 by IL-1 and TNF in human articular chondrocytes

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Inhibition of interleukin-1-stimulated MAP kinases, activating protein-1 (AP-1) and nuclear factor kappa B (NF-kappa B) transcription factors down-regulates matrix metalloproteinase gene expression in articular chondrocytes

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Regulation of the chondrocyte phenotype by beta-catenin

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Wnt-7a causes loss of differentiated phenotype and inhibits apoptosis of articular chondrocytes via different mechanisms

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10.1038/LABINVEST.3700747

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

ResearchGate publication ID

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