Inhibition of glutathione synthesis can enhance cycloheximide-induced protection of developing neurons against axotomy (Q48602934)

13 February 2018

title

Inhibition of glutathione synthesis can enhance cycloheximide-induced protection of developing neurons against axotomy (English)

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13 February 2018

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13 February 2018

author name string

Clarke PG

series ordinal

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13 February 2018

publication date

1 September 1997

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13 February 2018

number of pages

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Developmental Brain Research

inferred from page(s)

published in

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13 February 2018

volume

102

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13 February 2018

issue

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13 February 2018

page(s)

285-290

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13 February 2018

Limits to the dependence of developing neurons on protein synthesis in their axonal target territory

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Axotomy-induced retinal ganglion cell death in development: its time-course and its diminution by antioxidants

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Neuronal death during development in the isthmo-optic nucleus of the chick: sustaining role of afferents from the tectum

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At least two mechanisms are involved in the death of retinal ganglion cells following target ablation in neonatal rats

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7 January 2021

Neuroprotective action of cycloheximide involves induction of bcl-2 and antioxidant pathways

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Superoxide dismutase delays neuronal apoptosis: a role for reactive oxygen species in programmed neuronal death

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7 January 2021

Inhibition of lipid peroxidation attenuates axotomy-induced apoptotic degeneration of facial motor neurons in neonatal rats

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7 January 2021

The effect of cycloheximide and ganglioside GM1 on the viability of retinotectally projecting ganglion cells following ablation of the superior colliculus in neonatal rats

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The effects of early tectal lesions on development in the retinal ganglion cell layer of chick embryos

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Molecular mechanisms of developmental neuronal death

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A simple technique to determine glutathione (GSH) levels and synthesis in ocular tissues as GSH-bimane adduct: application to normal and galactosemic guinea-pigs.

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Thiol agents and Bcl-2 identify an alphavirus-induced apoptotic pathway that requires activation of the transcription factor NF-kappa B.

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Biochemical characterization of programmed cell death in NGF-deprived sympathetic neurons

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Inhibitors of protein synthesis and RNA synthesis prevent neuronal death caused by nerve growth factor deprivation

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Glutathione biosynthesis and its inhibition.

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Cell death during development of the nervous system

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Naturally occurring and induced neuronal death in the chick embryo in vivo requires protein and RNA synthesis: Evidence for the role of cell death genes

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Intracerebroventricular administration of L-buthionine sulfoximine: a method for depleting brain glutathione

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Immunocytochemical evidence for the presence of high levels of reduced glutathione in radial glial cells and horizontal cells in the rabbit retina

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Glutathiione system of human retina: Enzymatic conjugation of lipid peroxidation products

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Apoptotic cell death induced by optic nerve lesion in the neonatal rat.

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Apoptotic death in an in vitro model of neuronal oxidative stress

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Macromolecular synthesis inhibitors prevent oxidative stress-induced apoptosis in embryonic cortical neurons by shunting cysteine from protein synthesis to glutathione.

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Survival factor-insensitive generation of reactive oxygen species induced by serum deprivation in neuronal cells

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A specific, sensitive, and rapid method for the determination of glutathione and its application in ocular tissues

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DNA adducts and chronic degenerative disease. Pathogenetic relevance and implications in preventive medicine.

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Redox regulation of transcriptional activators

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Buthionine sulfoximine-induced glutathione depletion. Its effect on antioxidants, lipid peroxidation and calcium homeostasis in the lung

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The physiological consequences of glutathione variations.

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Glutathione oxidation in retina: effects on biochemical and electrical activities

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Cellular defenses against damage from reactive oxygen species

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Identifiers

DOI

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13 February 2018

1 reference

13 February 2018