Expression of amino-terminally truncated PrP in the mouse leading to ataxia and specific cerebellar lesions (Q48479400)

11 February 2018

title

Expression of amino-terminally truncated PrP in the mouse leading to ataxia and specific cerebellar lesions (English)

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11 February 2018

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11 February 2018

Christian von Mering

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11 February 2018

Adriano Aguzzi

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11 February 2018

Sebastian Brandner

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ORCID Public Data File 2021

author name string

Shmerling D

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11 February 2018

Hegyi I

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11 February 2018

Fischer M

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11 February 2018

Blättler T

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11 February 2018

Brandner S

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11 February 2018

Rülicke T

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11 February 2018

Flechsig E

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11 February 2018

Cozzio A

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11 February 2018

Hangartner C

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11 February 2018

Weissmann C

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11 February 2018

language of work or name

English

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publication date

1 April 1998

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11 February 2018

published in

Cell

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11 February 2018

volume

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11 February 2018

issue

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11 February 2018

page(s)

203-214

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11 February 2018

Nearly ubiquitous tissue distribution of the scrapie agent precursor protein

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Normal host prion protein necessary for scrapie-induced neurotoxicity

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Factors affecting the efficiency of introducing foreign DNA into mice by microinjecting eggs

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B lymphocytes and neuroinvasion

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Normal development and behaviour of mice lacking the neuronal cell-surface PrP protein

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Truncated forms of the human prion protein in normal brain and in prion diseases

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Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

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Hippocampal slices from prion protein null mice: disrupted Ca(2+)-activated K+ currents.

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7 January 2021

Prion protein is necessary for normal synaptic function

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7 January 2021

Structure of the recombinant full-length hamster prion protein PrP(29-231): the N terminus is highly flexible

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Prion protein (PrP) with amino-proximal deletions restoring susceptibility of PrP knockout mice to scrapie.

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Various rat adult tissues express only one major mRNA species from the glyceraldehyde-3-phosphate-dehydrogenase multigenic family

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7 January 2021

Processing of a cellular prion protein: identification of N- and C-terminal cleavage sites.

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Patch-clamp analysis of synaptic transmission to cerebellar purkinje cells of prion protein knockout mice

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Serial transmission in rodents of neurodegeneration from transgenic mice expressing mutant prion protein

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Mice deficient for prion protein exhibit normal neuronal excitability and synaptic transmission in the hippocampus

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7 January 2021

129/Ola mice carrying a null mutation in PrP that abolishes mRNA production are developmentally normal

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7 January 2021

Complementary hydropathy identifies a cellular prion protein receptor

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Targeting of transforming growth factor-alpha expression to pituitary lactotrophs in transgenic mice results in selective lactotroph proliferation and adenomas

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Heritable disorder resembling neuronal storage disease in mice expressing prion protein with deletion of an α-helix

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A cellular gene encodes scrapie PrP 27-30 protein

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Primary degeneration of the granular layer of the cerebellum. A study of 14 patients and review of the literature

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Metal-chelate affinity chromatography

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Novel proteinaceous infectious particles cause scrapie

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7 January 2021

The human 37-kDa laminin receptor precursor interacts with the prion protein in eukaryotic cells

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7 January 2021

NMR characterization of the full‐length recombinant murine prion protein, mPrP(23–231)

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7 January 2021

Loss of cerebellar Purkinje cells in aged mice homozygous for a disrupted PrP gene

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7 January 2021

DNA sequencing with chain-terminating inhibitors

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7 January 2021

Interactions between wild-type and mutant prion proteins modulate neurodegeneration in transgenic mice

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7 January 2021

Altered circadian activity rhythms and sleep in mice devoid of prion protein

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7 January 2021

PrP effects clarified

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7 January 2021

Degeneration of skeletal muscle, peripheral nerves, and the central nervous system in transgenic mice overexpressing wild-type prion proteins

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7 January 2021

Rescue of neurophysiological phenotype seen in PrP null mice by transgene encoding human prion protein

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7 January 2021

Genetic manipulation of mammals and its application in reproductive biology

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7 January 2021

Identification of candidate proteins binding to prion protein

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7 January 2021

Increased expression of neurofilament subunit NF-L produces morphological alterations that resemble the pathology of human motor neuron disease

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7 January 2021

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Identifiers

DOI

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11 February 2018

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11 February 2018