Delayed neuronal loss after administration of intracerebroventricular kainic acid to preweanling rats (Q48285347)

10 February 2018

title

Delayed neuronal loss after administration of intracerebroventricular kainic acid to preweanling rats (English)

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10 February 2018

author name string

Montgomery EM

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10 February 2018

Bardgett ME

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10 February 2018

Lall B

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10 February 2018

Csernansky CA

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10 February 2018

Csernansky JG

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10 February 2018

publication date

1 January 1999

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10 February 2018

number of pages

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Developmental Brain Research

inferred from page(s)

published in

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10 February 2018

volume

112

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10 February 2018

issue

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10 February 2018

page(s)

107-116

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10 February 2018

Emerging principles of intrinsic hippocampal organization

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Kainic acid decreases hippocampal neuronal number and increases dopamine receptor binding in the nucleus accumbens: an animal model of schizophrenia

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Kainic acid lesions enhance locomotor responses to novelty, saline, amphetamine, and MK-801.

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Ontogenetic development of kainate neurotoxicity: correlates with glutamatergic innervation

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Intrahippocampal injection of kainic acid produces significant pyramidal cell loss in neonatal rats

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Progressive neurodegeneration after intracerebroventricular kainic acid administration in rats: implications for schizophrenia?

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Altered gene expression in neurons during programmed cell death: identification of c-jun as necessary for neuronal apoptosis

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Evaluation of the mechanisms underlying the kainate-induced impairment of [3H]dopamine release in the rat striatum.

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Naturally occurring cell death in the subicular complex and hippocampus in the rat during development

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The heat-shock response

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Increased expression of mRNA encoding calbindin-D28K, the glucose-regulated proteins, or the 72 kDa heat-shock protein in three models of acute CNS injury

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Delayed cell death in the contralateral hippocampus following kainate injection into the CA3 subfield

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Induction of c-fos mRNA expression in an in vitro hippocampal slice model of adult rats after kainate but not gamma-aminobutyric acid or bicuculline treatment

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An immature mossy fiber innervation of hilar neurons may explain their resistance to kainate-induced cell death in 15-day-old rats

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Impaired learning and memory after kainic acid lesions of the striatum: a behavioral model of Huntington's disease

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7 January 2021

Cycloheximide prevents kainate-induced neuronal death and c-fos expression in adult rat brain.

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7 January 2021

Implications of normal brain development for the pathogenesis of schizophrenia

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Three-dimensional counting: an accurate and direct method to estimate numbers of cells in sectioned material

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Kainate and glutamate neurotoxicity in dependence on the postnatal development with special reference to hippocampal neurons

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Size, shape, and orientation of neurons in the left and right hippocampus: investigation of normal asymmetries and alterations in schizophrenia

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Identifiers

DOI

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10 February 2018

release_usahhqiq4zhx3dsutugvwiw6s4

Fatcat ID

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Fatcat

https://api.fatcat.wiki/v0/release/usahhqiq4zhx3dsutugvwiw6s4

24 November 2022

mapped directly with Wikidata item

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10 February 2018