Expression of Bcl-2, Bax and Caspase-3 in nerve tissues of rats chronically exposed to 2,5-hexanedione (Q48173499)

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English	Expression of Bcl-2, Bax and Caspase-3 in nerve tissues of rats chronically exposed to 2,5-hexanedione	scientific article

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17492505

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Expression of Bcl-2, Bax and Caspase-3 in nerve tissues of rats chronically exposed to 2,5-hexanedione (English)

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Ning Cui

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17492505

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Shanxia Li

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Xiulan Zhao

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17492505

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Tianliang Zhang

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Cuili Zhang

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Lihua Yu

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Zhengping Zhu

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Keqin Xie

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11 May 2007

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Neurochemical Research

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32

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9

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1566-1572

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GADD45 is induced in Alzheimer's disease, and protects against apoptosis in vitro

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Neuronal apoptosis induced by pharmacological concentrations of 3-hydroxykynurenine: characterization and protection by dantrolene and Bcl-2 overexpression

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Activation of CPP32 during apoptosis of neurons and astrocytes

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Apoptosis in the Nervous System: Morphological Features, Methods, Pathology, and Prevention

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Evidence of zinc protection against 2,5-hexanedione neurotoxicity: correlation of neurobehavioral testing with biomarkers of excretion.

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Total number and mean cell volume of neocortical neurons in rats exposed to 2,5-hexanedione with and without acetone

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Cytoskeletal changes induced by 2,5-hexanedione on developing human neurons in vitro

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2,5-Hexanedione and acrylamide produce reorganization of motoneuron perikarya.

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Expression of Fos, Jun, and Krox family proteins in Alzheimer's disease.

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Bcl-2, Bax and Bcl-x expression following kainic acid administration at convulsant doses in the rat.

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The effects of 2,5-hexanedione on the retina of albino rats

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Immunoreactivity for Bcl-2 protein within neurons in the Alzheimer's disease brain increases with disease severity

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2,5-Hexanedione induced apoptosis in cultured mouse DRG neurons

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Proapoptotic protein Bax heterodimerizes with Bcl-2 and homodimerizes with Bax via a novel domain (BH3) distinct from BH1 and BH2

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Induction of caspase-3-like protease may mediate delayed neuronal death in the hippocampus after transient cerebral ischemia.

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Acetone potentiation and influence on the reversibility of 2,5-hexanedione-induced neurotoxicity studied with behavioural and morphometric methods in rats

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Upregulation of the Anti-apoptotic Protein Bcl-2 May Be an Early Event in Neurodegeneration: Studies on Parkinson's and Incidental Lewy Body Disease

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Cell death mechanisms in Parkinson's disease

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Cytoskeletal effects of acrylamide and 2,5-hexanedione: selective aggregation of vimentin filaments

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Apoptosis and the nervous system

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Lack of evidence for the size principle of selective vulnerability of axons in toxic neuropathies. I. The effects of subcutaneous injections of 2,5-hexanedione on behavior and muscle spindle function

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Early spatial memory deficit induced by 2,5-hexanedione in the rat.

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Cell body remodeling during dying-back axonopathy: DRG changes during advanced disease

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BAX is required for neuronal death after trophic factor deprivation and during development

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gamma-diketone peripheral neuropathy. I. Quality morphometric analyses of axonal atrophy and swelling

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Toxicity and metabolism of the neurotoxic hexacarbons n-hexane, 2-hexanone, and 2,5-hexanedione

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bcl-2 protein is increased in the brain from parkinsonian patients

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Caspase-3: A vulnerability factor and final effector in apoptotic death of dopaminergic neurons in Parkinson's disease.

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Schwanm cell abnormalities in 2, 5-hexanedione neuropathy

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10.1007/S11064-007-9359-0

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17492505

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9 February 2018

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http://europepmc.org/abstract/MED/17492505

PubMed publication ID

17492505

1 reference

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PubMed publication ID

17492505

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9 February 2018

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http://europepmc.org/abstract/MED/17492505

 

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