Pathogenic Role for γδ T Cells in Autoimmune Anti-Myeloperoxidase Glomerulonephritis. (Q47734863)

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31 March 2020

title

Pathogenic Role for γδ T Cells in Autoimmune Anti-Myeloperoxidase Glomerulonephritis (English)

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31 March 2020

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31 March 2020

Joshua Daniel Ooi

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31 March 2020

Raymond Shim

series ordinal

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Dragana Odobasic

series ordinal

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Stephen Roger Holdsworth

series ordinal

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language of work or name

English

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publication date

27 September 2017

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31 March 2020

published in

Journal of Immunology

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volume

199

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issue

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page(s)

3042-3050

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Rapidly progressive crescentic glomerulonephritis

31 March 2020

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Th17 cells promote autoimmune anti-myeloperoxidase glomerulonephritis

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T cell mediated autoimmune glomerular disease in mice

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Glomerulonephritis Induced by Heterologous Anti-GBM Globulin as a Planted Foreign Antigen

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The immunodominant myeloperoxidase T-cell epitope induces local cell-mediated injury in antimyeloperoxidase glomerulonephritis.

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Mast cells contribute to peripheral tolerance and attenuate autoimmune vasculitis.

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Innate IL-17A-producing leukocytes promote acute kidney injury via inflammasome and Toll-like receptor activation

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

Identifiers

DOI

10.4049/JIMMUNOL.1602025

1 reference

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31 March 2020

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