Control of cell proliferation by Myc. (Q47721327)

5 February 2018

review article

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title

Control of cell proliferation by Myc. (English)

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5 February 2018

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5 February 2018

author name string

Bouchard C

series ordinal

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5 February 2018

Staller P

series ordinal

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5 February 2018

publication date

1 May 1998

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5 February 2018

published in

Trends in Cell Biology

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5 February 2018

volume

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5 February 2018

issue

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5 February 2018

page(s)

202-206

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5 February 2018

Inhibition of cell proliferation by the Mad1 transcriptional repressor

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Mnt, a novel Max-interacting protein is coexpressed with Myc in proliferating cells and mediates repression at Myc binding sites

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Histone deacetylases associated with the mSin3 corepressor mediate mad transcriptional repression

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Cell growth inhibition by the Mad/Max complex through recruitment of histone deacetylase activity

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Role for N-CoR and histone deacetylase in Sin3-mediated transcriptional repression

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Reciprocal regulation of adipogenesis by Myc and C/EBP alpha

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c-Myc represses transcription in vivo by a novel mechanism dependent on the initiator element and Myc box II

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Direct role for Myc in transcription initiation mediated by interactions with TFII-I

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YY1 and c-Myc associate in vivo in a manner that depends on c-Myc levels

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An alternative pathway for gene regulation by Myc

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Activation of cyclin-dependent kinases by Myc mediates induction of cyclin A, but not apoptosis

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Repression of c-Myc responsive genes in cycling cells causes G1 arrest through reduction of cyclin E/CDK2 kinase activity

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Identification of Positively and Negatively Acting Elements Regulating Expression of the E2F2 Gene in Response to Cell Growth Signals

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p21CIP1 and Cdc25A: competition between an inhibitor and an activator of cyclin-dependent kinases

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Cdk2-dependent phosphorylation of p27 facilitates its Myc-induced release from cyclin E/cdk2 complexes

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Cyclin E-CDK2 is a regulator of p27Kip1.

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Myc activation of cyclin E/Cdk2 kinase involves induction of cyclin E gene transcription and inhibition of p27(Kip1) binding to newly formed complexes

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Activation of c-Myc uncouples DNA replication from activation of G1-cyclin-dependent kinases

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Myc but not Fos rescue of PDGF signalling block caused by kinase-inactive Src.

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Cellular targets for activation by the E2F1 transcription factor include DNA synthesis- and G1/S-regulatory genes

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7 January 2021

pRB and p107/p130 are required for the regulated expression of different sets of E2F responsive genes.

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BIN1 is a novel MYC-interacting protein with features of a tumour suppressor

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Interaction of c-Myc with the pRb-related protein p107 results in inhibition of c-Myc-mediated transactivation

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Binding and suppression of the Myc transcriptional activation domain by p107.

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Mutual requirement of CDK4 and Myc in malignant transformation: evidence for cyclin D1/CDK4 and p16INK4A as upstream regulators of Myc.

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Myc and Ras collaborate in inducing accumulation of active cyclin E/Cdk2 and E2F.

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Ras signalling linked to the cell-cycle machinery by the retinoblastoma protein

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Raf1 interaction with Cdc25 phosphatase ties mitogenic signal transduction to cell cycle activation

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Cooperating oncogenes converge to regulate cyclin/cdk complexes.

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Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

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MYC abrogates p53-mediated cell cycle arrest in N-(phosphonacetyl)-L-aspartate-treated cells, permitting CAD gene amplification

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Cyclin E and c-Myc promote cell proliferation in the presence of p16INK4a and of hypophosphorylated retinoblastoma family proteins.

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7 January 2021

Cyclin E-induced S phase without activation of the pRb/E2F pathway.

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7 January 2021

Phosphorylation-dependent degradation of the cyclin-dependent kinase inhibitor p27Kip1

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7 January 2021

Induction of apoptosis in fibroblasts by c-myc protein

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7 January 2021

Suppression of c-Myc-induced apoptosis by Ras signalling through PI(3)K and PKB.

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7 January 2021

The PI 3-kinase/Akt signaling pathway delivers an anti-apoptotic signal.

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7 January 2021

Requirement for the CD95 Receptor-Ligand Pathway in c-Myc-Induced Apoptosis

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7 January 2021

Induction of TNF-sensitive cellular phenotype by c-Myc involves p53 and impaired NF-kappaB activation

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7 January 2021

10.1016/S0962-8924(98)01251-3

Identifiers

DOI

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5 February 2018

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5 February 2018