Correction of murine Rag1 deficiency by self-inactivating lentiviral vector-mediated gene transfer. (Q45870373)

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2 February 2020

title

Correction of murine Rag1 deficiency by self-inactivating lentiviral vector-mediated gene transfer (English)

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K Pike-Overzet

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M Rodijk

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Y-Y Ng

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M R M Baert

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A Schambach

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F Zhang

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S Hacein-Bey-Abina

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A C Lankester

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R G M Bredius

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G J A Driessen

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A J Thrasher

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C Baum

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F J T Staal

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publication date

27 May 2011

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volume

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issue

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page(s)

1471-1483

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Primary immunodeficiency diseases: an update from the International Union of Immunological Societies Primary Immunodeficiency Diseases Classification Committee Meeting in Budapest, 2005

2 February 2020

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In vivo gene delivery and stable transduction of nondividing cells by a lentiviral vector

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Multiply attenuated lentiviral vector achieves efficient gene delivery in vivo

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Self-inactivating retroviral vectors designed for transfer of whole genes into mammalian cells

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Transcription start regions in the human genome are favored targets for MLV integration

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RAG-1-deficient mice have no mature B and T lymphocytes

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A ubiquitous chromatin opening element (UCOE) confers resistance to DNA methylation-mediated silencing of lentiviral vectors

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Identification of two catalytic residues in RAG1 that define a single active site within the RAG1/RAG2 protein complex.

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7 January 2021

Identical mutations in RAG1 or RAG2 genes leading to defective V(D)J recombinase activity can cause either T-B-severe combined immune deficiency or Omenn syndrome

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Codon and mRNA Sequence Optimization of Microdystrophin Transgenes Improves Expression and Physiological Outcome in Dystrophic mdx Mice Following AAV2/8 Gene Transfer

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7 January 2021

Identifiers

DOI

10.1038/LEU.2011.106

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:21617701%20AND%20SRC:MED&resulttype=core&format=json

Dimensions Publication ID

2 February 2020

1018437224

0 references

1 reference

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