Inter-mitochondrial complementation: Mitochondria-specific system preventing mice from expression of disease phenotypes by mutant mtDNA. (Q45878904)

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English	Inter-mitochondrial complementation: Mitochondria-specific system preventing mice from expression of disease phenotypes by mutant mtDNA.	scientific article

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11479626

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17 December 2017

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http://europepmc.org/abstract/MED/11479626

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Inter-mitochondrial complementation: Mitochondria-specific system preventing mice from expression of disease phenotypes by mutant mtDNA. (English)

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11479626

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17 December 2017

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mitochondrion

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phenotype

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Nakada K

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1

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11479626

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Inoue K

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2

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Isobe K

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Ogura A

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Goto YI

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Nonaka I

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Hayashi JI

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English

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1 August 2001

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Nature Medicine

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7

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8

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934-940

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cites work

Molecular genetic aspects of human mitochondrial disorders

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Mitochondrial diseases in man and mouse

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Dilated cardiomyopathy and atrioventricular conduction blocks induced by heart-specific inactivation of mitochondrial DNA gene expression.

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Mice with only rat mtDNA are required as models of mitochondrial diseases

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Isolation of mitochondrial DNA-less mouse cell lines and their application for trapping mouse synaptosomal mitochondrial DNA with deletion mutations

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Generation of mice with mitochondrial dysfunction by introducing mouse mtDNA carrying a deletion into zygotes

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Complementation of mutant and wild-type human mitochondrial DNAs coexisting since the mutation event and lack of complementation of DNAs introduced separately into a cell within distinct organelles

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Distribution of wild-type and common deletion forms of mtDNA in normal and respiration-deficient muscle fibers from patients with mitochondrial myopathy

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Introduction of disease-related mitochondrial DNA deletions into HeLa cells lacking mitochondrial DNA results in mitochondrial dysfunction

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Human cells are protected from mitochondrial dysfunction by complementation of DNA products in fused mitochondria

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Human mitochondria and mitochondrial genome function as a single dynamic cellular unit

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Functional integrity of mitochondrial genomes in human platelets and autopsied brain tissues from elderly patients with Alzheimer's disease

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Nondroplet ultrastructural demonstration of cytochrome oxidase activity with a polymerizing osmiophilic reagent, diaminobenzidine (DAB).

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Cytochrome C oxidase deficiency in two siblings with leigh encephalomyelopathy

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Tissue specificity in cytochrome c oxidase deficient myopathy

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Identifiers

DOI

10.1038/90976

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Europe PubMed Central

PubMed publication ID

11479626

retrieved

17 December 2017

reference URL

http://europepmc.org/abstract/MED/11479626

Dimensions Publication ID

1020556775

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PubMed publication ID

11479626

1 reference

stated in

Europe PubMed Central

PubMed publication ID

11479626

retrieved

17 December 2017

reference URL

http://europepmc.org/abstract/MED/11479626

 

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