Association of common single-nucleotide polymorphisms in innate immune genes with differences in TLR-induced cytokine production in neonates. (Q45342887)

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English	Association of common single-nucleotide polymorphisms in innate immune genes with differences in TLR-induced cytokine production in neonates.	scientific article

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25 February 2020

Association of common single-nucleotide polymorphisms in innate immune genes with differences in TLR-induced cytokine production in neonates (English)

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25 February 2020

toll-like receptor

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single-nucleotide polymorphism

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3

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25 February 2020

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25 February 2020

Stuart E Turvey

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25 February 2020

Edgardo S Fortuno

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25 February 2020

Tobias Kollmann

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25 February 2020

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P Cho

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25 February 2020

S J Tebbutt

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25 February 2020

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7 March 2013

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25 February 2020

Genes and Immunity

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25 February 2020

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25 February 2020

4

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25 February 2020

199-211

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25 February 2020

Pathogen recognition and innate immunity

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The control of adaptive immune responses by the innate immune system

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Translating innate immunity into immunological memory: implications for vaccine development

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Toll-like Receptors and Their Crosstalk with Other Innate Receptors in Infection and Immunity

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Human TLRs 10 and 1 share common mechanisms of innate immune sensing but not signaling.

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Single nucleotide polymorphisms in innate immunity genes: abundant variation and potential role in complex human disease

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Innate immune function by Toll-like receptors: distinct responses in newborns and the elderly

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Neonatal innate TLR-mediated responses are distinct from those of adults.

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Human TLR10 is a functional receptor, expressed by B cells and plasmacytoid dendritic cells, which activates gene transcription through MyD88

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The L412F variant of Toll-like receptor 3 (TLR3) is associated with cutaneous candidiasis, increased susceptibility to cytomegalovirus, and autoimmunity

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