Expression of mutated huntingtin fragment in the putamen is sufficient to produce abnormal movement in non-human primates (Q45305166)

11 December 2017

title

Expression of mutated huntingtin fragment in the putamen is sufficient to produce abnormal movement in non-human primates (English)

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11 December 2017

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11 December 2017

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11 December 2017

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11 December 2017

Nicole Déglon

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Philippe Hantraye

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11 December 2017

Stéphane Palfi

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11 December 2017

Caroline Jan

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11 December 2017

Masahiro Shin

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11 December 2017

Françoise Condé

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11 December 2017

Xiao-Jiang Li

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11 December 2017

Patrick Aebischer

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11 December 2017

publication date

1 May 2007

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11 December 2017

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11 December 2017

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11 December 2017

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11 December 2017

page(s)

1444-1451

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A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes.

11 December 2017

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Chronic mitochondrial energy impairment produces selective striatal degeneration and abnormal choreiform movements in primates

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Chronic 3-nitropropionic acid treatment in baboons replicates the cognitive and motor deficits of Huntington's disease

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Fetal striatal allografts reverse cognitive deficits in a primate model of Huntington disease

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Restoration of cognitive and motor functions by ciliary neurotrophic factor in a primate model of Huntington's disease.

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A primate model of Huntington's disease: behavioral and anatomical studies of unilateral excitotoxic lesions of the caudate-putamen in the baboon

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Selective putaminal excitotoxic lesions in non-human primates model the movement disorder of Huntington disease.

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Lentiviral-mediated delivery of mutant huntingtin in the striatum of rats induces a selective neuropathology modulated by polyglutamine repeat size, huntingtin expression levels, and protein length.

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Early and reversible neuropathology induced by tetracycline-regulated lentiviral overexpression of mutant huntingtin in rat striatum

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Aggregation of huntingtin in neuronal intranuclear inclusions and dystrophic neurites in brain

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Evidence for apoptotic cell death in Huntington disease and excitotoxic animal models.

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Formation of neuronal intranuclear inclusions underlies the neurological dysfunction in mice transgenic for the HD mutation

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Amino-terminal fragments of mutant huntingtin show selective accumulation in striatal neurons and synaptic toxicity

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Long glutamine tracts cause nuclear localization of a novel form of huntingtin in medium spiny striatal neurons in HdhQ92 and HdhQ111 knock-in mice

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7 January 2021

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Organization of corticostriatal motor inputs in monkey putamen

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Replication of the neurochemical characteristics of Huntington's disease by quinolinic acid

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7 January 2021

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7 January 2021

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7 January 2021

Self-inactivating lentiviral vectors with enhanced transgene expression as potential gene transfer system in Parkinson's disease

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7 January 2021

Complete and long-term rescue of lesioned adult motoneurons by lentiviral-mediated expression of glial cell line-derived neurotrophic factor in the facial nucleus.

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7 January 2021

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7 January 2021

Delayed onset of progressive dystonia following subacute 3-nitropropionic acid treatment in Cebus apella monkeys

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7 January 2021

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7 January 2021

Nuclear and neuropil aggregates in Huntington's disease: relationship to neuropathology.

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7 January 2021

The use of antiavidin antibody and avidin-biotin-peroxidase complex in immunoperoxidase technics

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7 January 2021

cFos immunoreactivity is enhanced with biotin amplification

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7 January 2021

Identifiers

DOI

10.1038/SJ.MT.6300185

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11 December 2017

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