The influence of age on apoptotic and other mechanisms of cell death after cerebral hypoxia-ischemia. (Q45183594)

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6 January 2020

title

The influence of age on apoptotic and other mechanisms of cell death after cerebral hypoxia-ischemia (English)

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6 January 2020

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6 January 2020

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6 January 2020

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6 January 2020

Zhu C

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6 January 2020

Xu F

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6 January 2020

Bahr BA

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6 January 2020

Shibata M

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6 January 2020

Uchiyama Y

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6 January 2020

publication date

1 February 2005

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Cell Death & Differentiation

6 January 2020

published in

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6 January 2020

volume

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issue

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6 January 2020

page(s)

162-176

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Involvement of caspase-3 in cell death after hypoxia-ischemia declines during brain maturation

6 January 2020

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Influence of age on the cerebral lesions in an immature rat model of cerebral hypoxia-ischemia: a light microscopic study

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The influence of immaturity on hypoxic‐ischemic brain damage in the rat

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Early Neurodegeneration after Hypoxia-Ischemia in Neonatal Rat Is Necrosis while Delayed Neuronal Death Is Apoptosis

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Pathogenesis of hippocampal neuronal death after hypoxia-ischemia changes during brain development

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Synergistic activation of caspase-3 by m-calpain after neonatal hypoxia-ischemia: a mechanism of "pathological apoptosis"?

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Alteration of apoptotic protease-activating factor-1 (APAF-1)-dependent apoptotic pathway during development of rat brain and liver

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Blockade of NMDA receptors and apoptotic neurodegeneration in the developing brain

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Correlation Between Caspase-3 Activation and Three Different Markers of DNA Damage in Neonatal Cerebral Hypoxia-Ischemia

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Immature and mature cortical neurons engage different apoptotic mechanisms involving caspase-3 and the mitogen-activated protein kinase pathway

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Caspase-3 activation after neonatal rat cerebral hypoxia-ischemia

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Caspase inhibitor affords neuroprotection with delayed administration in a rat model of neonatal hypoxic-ischemic brain injury

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X-linked inhibitor of apoptosis (XIAP) protein protects against caspase activation and tissue loss after neonatal hypoxia-ischemia

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Role of caspase-3 activation in cerebral ischemia-induced neurodegeneration in adult and neonatal brain

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Nuclear condensation and fragmentation following cerebral hypoxia-ischemia occurs more frequently in immature than older rats

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Marked age-dependent neuroprotection by brain-derived neurotrophic factor against neonatal hypoxic-ischemic brain injury

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Prevention of hypoxic-ischemic damage with dexamethasone is dependent on age and not influenced by fasting

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Activation of ERK1/2 after neonatal rat cerebral hypoxia-ischaemia

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Involvement of apoptosis-inducing factor in neuronal death after hypoxia-ischemia in the neonatal rat brain

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Nuclear translocation of apoptosis-inducing factor after focal cerebral ischemia

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Irradiation-induced progenitor cell death in the developing brain is resistant to erythropoietin treatment and caspase inhibition

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Calpastatin is up-regulated in response to hypoxia and is a suicide substrate to calpain after neonatal cerebral hypoxia-ischemia

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Fodrin degradation and subcellular distribution of calpains after neonatal rat cerebral hypoxic-ischemia

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Nitrosylation precedes caspase-3 activation and translocation of apoptosis-inducing factor in neonatal rat cerebral hypoxia-ischaemia

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Differential expression of apoptotic protease-activating factor-1 and caspase-3 genes and susceptibility to apoptosis during brain development and after traumatic brain injury.

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Protective effects of moderate hypothermia after neonatal hypoxia-ischemia: short- and long-term outcome

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7 January 2021

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7 January 2021

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7 January 2021

Intrauterine hypoxia-ischemia alters nitric oxide synthase expression and activity in fetal and neonatal rat brains

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7 January 2021

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7 January 2021

Subcellular distribution of calcium and ultrastructural changes after cerebral hypoxia-ischemia in immature rats

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7 January 2021

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An absolute method for protein determination based on difference in absorbance at 235 and 280 nm

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7 January 2021

PARP-1 gene disruption in mice preferentially protects males from perinatal brain injury.

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7 January 2021

Frequent episodes of brief ischemia sensitize the fetal sheep brain to neuronal loss and induce striatal injury.

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7 January 2021

Identifiers

DOI

10.1038/SJ.CDD.4401545

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Dimensions Publication ID

6 January 2020

1000836968

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24 November 2022

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