Number of oligodendrocyte progenitors recruited to the lesioned spinal cord is modulated by the levels of the cell cycle regulatory protein p27Kip-1. (Q45097238)

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scientific article published in January 2005

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English	Number of oligodendrocyte progenitors recruited to the lesioned spinal cord is modulated by the levels of the cell cycle regulatory protein p27Kip-1.	scientific article published in January 2005

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

9 December 2017

Number of oligodendrocyte progenitors recruited to the lesioned spinal cord is modulated by the levels of the cell cycle regulatory protein p27Kip-1. (English)

1 reference

Europe PubMed Central

PubMed publication ID

9 December 2017

oligodendrocyte

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inferred from title

David P Crockett

1

object named as

David P Crockett

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Europe PubMed Central

PubMed publication ID

9 December 2017

author name string

Mark Burshteyn

2

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Europe PubMed Central

PubMed publication ID

9 December 2017

Corina Garcia

3

1 reference

Europe PubMed Central

PubMed publication ID

9 December 2017

Michela Muggironi

4

1 reference

Europe PubMed Central

PubMed publication ID

9 December 2017

Patrizia Casaccia-Bonnefil

5

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Europe PubMed Central

PubMed publication ID

9 December 2017

publication date

1 January 2005

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Europe PubMed Central

PubMed publication ID

9 December 2017

1 reference

Europe PubMed Central

PubMed publication ID

9 December 2017

49

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Europe PubMed Central

PubMed publication ID

9 December 2017

2

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Europe PubMed Central

PubMed publication ID

9 December 2017

301-308

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Europe PubMed Central

PubMed publication ID

9 December 2017

The origin of remyelinating cells in the central nervous system

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Identification of the adult resting progenitor cell by autoradiographic tracking of oligodendrocyte precursors in experimental CNS demyelination

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7 January 2021

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Oligodendrocyte precursor differentiation is perturbed in the absence of the cyclin-dependent kinase inhibitor p27Kip1

1 reference

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Generation of oligodendroglial progenitors in acute inflammatory demyelinating lesions of the rat brain stem is associated with demyelination rather than inflammation

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p27Kip1 alters the response of cells to mitogen and is part of a cell-intrinsic timer that arrests the cell cycle and initiates differentiation.

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Why does remyelination fail in multiple sclerosis?

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Endogenous Progenitors Remyelinate Demyelinated Axons in the Adult CNS

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Voltage-activated K+ channels and membrane depolarization regulate accumulation of the cyclin-dependent kinase inhibitors p27(Kip1) and p21(CIP1) in glial progenitor cells.

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Activation and proliferation of endogenous oligodendrocyte precursor cells during ethidium bromide-induced demyelination

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Platelet-derived growth factor-alpha receptor-positive oligodendroglia are frequent in multiple sclerosis lesions

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Impaired remyelination and depletion of oligodendrocyte progenitors does not occur following repeated episodes of focal demyelination in the rat central nervous system

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In vivo proliferation of oligodendrocyte progenitors expressing PDGFalphaR during early remyelination

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The response of adult oligodendrocyte progenitors to demyelination in EAE.

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The age-related decrease in CNS remyelination efficiency is attributable to an impairment of both oligodendrocyte progenitor recruitment and differentiation.

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Cell cycle arrest induced by ectopic expression of p27 is not sufficient to promote oligodendrocyte differentiation

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Changes in cyclin-dependent kinase 2 and p27kip1 accompany glial cell differentiation of central glia-4 cells

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7 January 2021

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Identifiers

10.1002/GLIA.20111

1 reference

Europe PubMed Central

PubMed publication ID

9 December 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

9 December 2017

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