Monoclonality and surface lesion-specific microsatellite alterations in premalignant and malignant neoplasia of uterine cervix: a local field effect of genomic instability and clonal evolution (Q44519800)

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scientific article published in February 1999

Language	Label	Description	Also known as
English	Monoclonality and surface lesion-specific microsatellite alterations in premalignant and malignant neoplasia of uterine cervix: a local field effect of genomic instability and clonal evolution	scientific article published in February 1999

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

3 December 2017

Monoclonality and surface lesion-specific microsatellite alterations in premalignant and malignant neoplasia of uterine cervix: a local field effect of genomic instability and clonal evolution (English)

1 reference

Europe PubMed Central

PubMed publication ID

3 December 2017

1 reference

based on heuristic

inferred from title

author name string

Chu TY

1

1 reference

Europe PubMed Central

PubMed publication ID

3 December 2017

Shen CY

2

1 reference

Europe PubMed Central

PubMed publication ID

3 December 2017

Lee HS

3

1 reference

Europe PubMed Central

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3 December 2017

Liu HS

4

1 reference

Europe PubMed Central

PubMed publication ID

3 December 2017

language of work or name

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based on heuristic

inferred from title

publication date

1 February 1999

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Europe PubMed Central

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3 December 2017

Genes, Chromosomes and Cancer

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Europe PubMed Central

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3 December 2017

24

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Europe PubMed Central

PubMed publication ID

3 December 2017

2

1 reference

Europe PubMed Central

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3 December 2017

127-134

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Europe PubMed Central

PubMed publication ID

3 December 2017

Clues to the pathogenesis of familial colorectal cancer

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Identification and assessment of known and novel human papillomaviruses by polymerase chain reaction amplification, restriction fragment length polymorphisms, nucleotide sequence, and phylogenetic algorithms

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HPV-associated cervical cancers show frequent allelic loss at 3p14 but no apparent aberration of FHIT mRNA

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A genetic model for colorectal tumorigenesis

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Multiple-mutation theory of carcinogenesis.

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Oxidative mechanisms in carcinogenesis

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Microsatellites for linkage analysis of genetic traits

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Genomic instability in neoplasia.

1 reference

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p53 tumor suppressor gene status and the degree of genomic instability in sporadic colorectal cancers

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Natural history of cervical neoplasia: overview and update

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Genetics of human cancer

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Consistent chromosome 3p deletion and loss of heterozygosity in renal cell carcinoma

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Genetic alterations at 5p15: a potential marker for progression of precancerous lesions of the uterine cervix

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The clonal evolution of tumor cell populations

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The FHIT gene, spanning the chromosome 3p14.2 fragile site and renal carcinoma-associated t(3;8) breakpoint, is abnormal in digestive tract cancers

1 reference

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Protein expression and functional analysis of the FHIT gene in human tumor cells

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21 January 2018

Variable FHIT transcripts in non-neoplastic tissues

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21 January 2018

Association between human papillomavirus type and clonal status of cervical squamous intraepithelial lesions

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21 January 2018

The FHIT gene 3p14.2 is abnormal in lung cancer.

1 reference

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21 January 2018

NormalFHIT transcripts in renal cell cancer- and lung cancer-derived cell lines, including a cell line with a homozygous deletion in the FRA3B region

1 reference

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Association of human papillomavirus types 16 and 18 E6 proteins with p53.

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FRA3B extends over a broad region and contains a spontaneous HPV16 integration site: direct evidence for the coincidence of viral integration sites and fragile sites

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Deletion of a DNA sequence at the chromosomal region 3p21 in all major types of lung cancer

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Genomic deletion and p53 inactivation in cervical carcinoma

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21 January 2018

Identifiers

10.1002/(SICI)1098-2264(199902)24:2<127::AID-GCC5>3.0.CO;2-8

1 reference

Europe PubMed Central

PubMed publication ID

3 December 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

3 December 2017

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