Possible involvement of both mitochondria- and endoplasmic reticulum-dependent caspase pathways in rotenone-induced apoptosis in human neuroblastoma SH-SY5Y cells (Q44195124)

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scientific article published in November 2002

Language	Label	Description	Also known as
English	Possible involvement of both mitochondria- and endoplasmic reticulum-dependent caspase pathways in rotenone-induced apoptosis in human neuroblastoma SH-SY5Y cells	scientific article published in November 2002

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

Possible involvement of both mitochondria- and endoplasmic reticulum-dependent caspase pathways in rotenone-induced apoptosis in human neuroblastoma SH-SY5Y cells (English)

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

endoplasmic reticulum

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inferred from title

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6

object named as

Shun Shimohama

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

author name string

Yoshihisa Kitamura

1

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Europe PubMed Central

PubMed publication ID

30 November 2017

Masatoshi Inden

2

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Europe PubMed Central

PubMed publication ID

30 November 2017

Atsushi Miyamura

3

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Europe PubMed Central

PubMed publication ID

30 November 2017

Jun-ichi Kakimura

4

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Europe PubMed Central

PubMed publication ID

30 November 2017

Takashi Taniguchi

5

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Europe PubMed Central

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30 November 2017

language of work or name

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publication date

1 November 2002

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Europe PubMed Central

PubMed publication ID

30 November 2017

number of pages

4

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Neuroscience Letters

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30 November 2017

333

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30 November 2017

1

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30 November 2017

25-28

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30 November 2017

Chronic systemic pesticide exposure reproduces features of Parkinson's disease

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Glycoxidation and oxidative stress in Parkinson disease and diffuse Lewy body disease

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From entropy to Duino

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Caspase-3: A vulnerability factor and final effector in apoptotic death of dopaminergic neurons in Parkinson's disease.

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Parkin suppresses unfolded protein stress-induced cell death through its E3 ubiquitin-protein ligase activity

1 reference

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7 January 2021

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An unfolded putative transmembrane polypeptide, which can lead to endoplasmic reticulum stress, is a substrate of Parkin

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Protective effects of the antiparkinsonian drugs talipexole and pramipexole against 1-methyl-4-phenylpyridinium-induced apoptotic death in human neuroblastoma SH-SY5Y cells.

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Nitric oxide donor-induced p53-sensitive cell death is enhanced by Bcl-2 reduction in human neuroblastoma cells

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The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

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Chronic Parkinsonism in humans due to a product of meperidine-analog synthesis

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Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

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An endoplasmic reticulum stress-specific caspase cascade in apoptosis. Cytochrome c-independent activation of caspase-9 by caspase-12

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Caspase-12 mediates endoplasmic-reticulum-specific apoptosis and cytotoxicity by amyloid-beta

1 reference

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Coupling endoplasmic reticulum stress to the cell death program. Mechanism of caspase activation

1 reference

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based on heuristic

inferred from DOI database lookup

Coupling endoplasmic reticulum stress to the cell death program. An Apaf-1-independent intrinsic pathway

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based on heuristic

inferred from DOI database lookup

Mitochondrial complex I deficiency in Parkinson's disease

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Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

1 reference

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based on heuristic

inferred from DOI database lookup

Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

1 reference

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based on heuristic

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Identifiers

10.1016/S0304-3940(02)00964-3

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

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