The TLR4/TRIF-Mediated Activation of NLRP3 Inflammasome Underlies Endotoxin-Induced Liver Injury in Mice (Q33996553)

30 July 2017

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title

The TLR4/TRIF-Mediated Activation of NLRP3 Inflammasome Underlies Endotoxin-Induced Liver Injury in Mice (English)

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30 July 2017

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Hiroko Tsutsui

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30 July 2017

Michiko Imamura

series ordinal

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30 July 2017

Jiro Fujimoto

series ordinal

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30 July 2017

Kenji Nakanishi

series ordinal

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30 July 2017

publication date

16 June 2010

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Gastroenterology research and practice

30 July 2017

published in

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30 July 2017

volume

2010

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30 July 2017

page(s)

641865

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Creative Commons Attribution 3.0 Unported

30 July 2017

start time

1 January 2010

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April 2022 Public Data File from Crossref

copyrighted

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April 2022 Public Data File from Crossref

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12 December 2020

Lipopolysaccharide-induced IL-18 secretion from murine Kupffer cells independently of myeloid differentiation factor 88 that is critically involved in induction of production of IL-12 and IL-1beta

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Disseminated intravascular coagulation

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IL-18-deficient mice are resistant to endotoxin-induced liver injury but highly susceptible to endotoxin shock

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12 December 2020

IL-12 up-regulates IL-18 receptor expression on T cells, Th1 cells, and B cells: synergism with IL-18 for IFN-gamma production

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IL-18 accounts for both TNF-alpha- and Fas ligand-mediated hepatotoxic pathways in endotoxin-induced liver injury in mice

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ASC is essential for LPS-induced activation of procaspase-1 independently of TLR-associated signal adaptor molecules

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12 December 2020

Importance of direct hepatocytolysis by liver macrophages in experimental fulminant hepatitis

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Caspase-1-independent, Fas/Fas ligand-mediated IL-18 secretion from macrophages causes acute liver injury in mice

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12 December 2020

Identifiers

DOI

10.1155/2010/641865

1 reference

30 July 2017

1 reference

30 July 2017

1 reference