Differential regulation of collapsin response mediator protein 2 (CRMP2) phosphorylation by GSK3ß and CDK5 following traumatic brain injury. (Q33671879)

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English	Differential regulation of collapsin response mediator protein 2 (CRMP2) phosphorylation by GSK3ß and CDK5 following traumatic brain injury.	scientific article

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20 March 2020

Differential regulation of collapsin response mediator protein 2 (CRMP2) phosphorylation by GSK3ß and CDK5 following traumatic brain injury (English)

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20 March 2020

traumatic brain injury

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phosphorylation

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based on heuristic

inferred from title

5

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20 March 2020

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Sarah M Wilson

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20 March 2020

Seul Ki Yeon

2

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20 March 2020

Xiao-Fang Yang

3

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20 March 2020

Ki Duk Park

4

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28 May 2014

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20 March 2020

Frontiers in Cellular Neuroscience

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20 March 2020

8

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20 March 2020

135

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20 March 2020

Effect of lacosamide on structural damage and functional recovery after traumatic brain injury in rats

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CRMP2 protein SUMOylation modulates NaV1.7 channel trafficking

1 reference

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Lacosamide treatment following status epilepticus attenuates neuronal cell loss and alterations in hippocampal neurogenesis in a rat electrical status epilepticus model

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SUMOylation alters CRMP2 regulation of calcium influx in sensory neurons

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Prevention of posttraumatic axon sprouting by blocking collapsin response mediator protein 2-mediated neurite outgrowth and tubulin polymerization.

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Disruption of NMDAR-CRMP-2 signaling protects against focal cerebral ischemic damage in the rat middle cerebral artery occlusion model

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21 June 2018

VOLTAGE-GATED CALCIUM CHANNELS ARE NOT AFFECTED BY THE NOVEL ANTI-EPILEPTIC DRUG LACOSAMIDE.

1 reference

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Neuroprotection against traumatic brain injury by a peptide derived from the collapsin response mediator protein 2 (CRMP2)

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Suppression of inflammatory and neuropathic pain by uncoupling CRMP-2 from the presynaptic Ca²⁺ channel complex

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Why mesial temporal lobe epilepsy with hippocampal sclerosis is progressive: uncontrolled inflammation drives disease progression?

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21 June 2018

In silico docking and electrophysiological characterization of lacosamide binding sites on collapsin response mediator protein-2 identifies a pocket important in modulating sodium channel slow inactivation

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An atypical role for collapsin response mediator protein 2 (CRMP-2) in neurotransmitter release via interaction with presynaptic voltage-gated calcium channels

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21 June 2018

Semaphorin3A signaling mediated by Fyn-dependent tyrosine phosphorylation of collapsin response mediator protein 2 at tyrosine 32.

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NS21: re-defined and modified supplement B27 for neuronal cultures

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21 June 2018

Mesial temporal lobe epilepsy: pathogenesis, induced rodent models and lesions

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21 June 2018

The investigational anticonvulsant lacosamide selectively enhances slow inactivation of voltage-gated sodium channels.

1 reference

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21 June 2018

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21 June 2018

Lacosamide: a review of preclinical properties.

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21 June 2018

Distinct priming kinases contribute to differential regulation of collapsin response mediator proteins by glycogen synthase kinase-3 in vivo

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21 June 2018

Phosphorylation by Rho kinase regulates CRMP-2 activity in growth cones

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21 June 2018

Post-traumatic epilepsy: an overview

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21 June 2018

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21 June 2018

Tubulin and CRMP-2 complex is transported via Kinesin-1

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21 June 2018

GSK-3beta regulates phosphorylation of CRMP-2 and neuronal polarity.

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21 June 2018

GSK-3 phosphorylation of the Alzheimer epitope within collapsin response mediator proteins regulates axon elongation in primary neurons.

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A retrospective analysis of hippocampal pathology in human temporal lobe epilepsy: evidence for distinctive patient subcategories

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21 June 2018

CRMP-2 binds to tubulin heterodimers to promote microtubule assembly

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Phosphorylation of collapsin response mediator protein-2 by Rho-kinase. Evidence for two separate signaling pathways for growth cone collapse

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Cdk5-mediated phosphorylation of CRMP-2 enhances its interaction with CaV2.2.

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Posttraumatic epilepsy

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CaMKII phosphorylates collapsin response mediator protein 2 and modulates axonal damage during glutamate excitotoxicity.

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Identifiers

10.3389/FNCEL.2014.00135

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Europe PubMed Central

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20 March 2020

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Europe PubMed Central

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20 March 2020

PubMed publication ID

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Europe PubMed Central

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20 March 2020

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