Bax expression correlates with cellular drug sensitivity to doxorubicin, cyclophosphamide and chlorambucil but not fludarabine, cladribine or corticosteroids in B cell chronic lymphocytic leukemia (Q33183751)

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scientific article published in June 2002

Language	Label	Description	Also known as
English	Bax expression correlates with cellular drug sensitivity to doxorubicin, cyclophosphamide and chlorambucil but not fludarabine, cladribine or corticosteroids in B cell chronic lymphocytic leukemia	scientific article published in June 2002

Statements

scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

23 July 2017

Bax expression correlates with cellular drug sensitivity to doxorubicin, cyclophosphamide and chlorambucil but not fludarabine, cladribine or corticosteroids in B cell chronic lymphocytic leukemia (English)

1 reference

Europe PubMed Central

PubMed publication ID

23 July 2017

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chronic lymphocytic leukemia

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author name string

Bosanquet AG

1

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Europe PubMed Central

PubMed publication ID

23 July 2017

Sturm I

2

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Europe PubMed Central

PubMed publication ID

23 July 2017

Wieder T

3

1 reference

Europe PubMed Central

PubMed publication ID

23 July 2017

Essmann F

4

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Europe PubMed Central

PubMed publication ID

23 July 2017

Bosanquet MI

5

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Europe PubMed Central

PubMed publication ID

23 July 2017

Head DJ

6

1 reference

Europe PubMed Central

PubMed publication ID

23 July 2017

Dörken B

7

1 reference

Europe PubMed Central

PubMed publication ID

23 July 2017

Daniel PT

8

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Europe PubMed Central

PubMed publication ID

23 July 2017

publication date

1 June 2002

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Europe PubMed Central

PubMed publication ID

23 July 2017

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Europe PubMed Central

PubMed publication ID

23 July 2017

16

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Europe PubMed Central

PubMed publication ID

23 July 2017

6

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Europe PubMed Central

PubMed publication ID

23 July 2017

1035-1044

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Europe PubMed Central

PubMed publication ID

23 July 2017

Dissecting the pathways to death

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Chemosensitivity of B cell chronic lymphocytic leukemia and correlated expression of proteins regulating apoptosis, cell cycle and DNA repair.

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Chlorambucil resistance in B-cell chronic lymphocytic leukaemia is mediated through failed Bax induction and selection of high Bcl-2-expressing subclones

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Impaired BAX protein expression in breast cancer: mutational analysis of the BAX and the p53 gene

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Overexpression of the death-promoting gene bax-alpha which is downregulated in breast cancer restores sensitivity to different apoptotic stimuli and reduces tumor growth in SCID mice

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Induction of the death-promoting gene bax-alpha sensitizes cultured breast-cancer cells to drug-induced apoptosis

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Expression of the bcl-2 gene family in normal and malignant breast tissue: low bax-alpha expression in tumor cells correlates with resistance towards apoptosis

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Relapse in childhood acute lymphoblastic leukemia is associated with a decrease of the Bax/Bcl-2 ratio and loss of spontaneous caspase-3 processing in vivo

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Activation of caspase-8 in drug-induced apoptosis of B-lymphoid cells is independent of CD95/Fas receptor-ligand interaction and occurs downstream of caspase-3.

1 reference

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Response to cladribine in previously treated patients with chronic lymphocytic leukaemia identified by ex vivo assessment of drug sensitivity by DiSC assay.

1 reference

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Prognosis for fludarabine therapy of chronic lymphocytic leukaemia based on ex vivo drug response by DiSC assay.

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Methylprednisolone in advanced chronic lymphocytic leukaemia: rationale for, and effectiveness of treatment suggested by DiSC assay.

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Enhanced ex vivo drug sensitivity testing of chronic lymphocytic leukaemia using refined DiSC assay methodology

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Measurement of protein using bicinchoninic acid

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Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4

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Growth factors stimulate phosphorylation of CTP:phosphocholine cytidylyltransferase in HeLa cells.

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Ex vivo assessment of drug response by differential staining cytotoxicity (DiSC) assay suggests a biological basis for equality of chemotherapy irrespective of age for patients with chronic lymphocytic leukaemia

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Analysis of p53/BAX/p16(ink4a/CDKN2) in esophageal squamous cell carcinoma: high BAX and p16(ink4a/CDKN2) identifies patients with good prognosis

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In vitro chemosensitivity of chronic lymphocytic leukaemia to purine analogues--correlation with clinical course

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Bcl-2, Bax and p53 expression in B-CLL in relation to in vitro survival and clinical progression

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Regulation of clinical chemoresistance by bcl-2 and bax oncoproteins in B-cell chronic lymphocytic leukaemia

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Overexpression of caspase-3 restores sensitivity for drug-induced apoptosis in breast cancer cell lines with acquired drug resistance

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Management of chronic lymphocytic leukaemia

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Prediction of prognosis following fludarabine used as secondary therapy for chronic lymphocytic leukemia

1 reference

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Nucleotide requirements for the in vitro activation of the apoptosis protein-activating factor-1-mediated caspase pathway

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Induction of an apoptotic program in cell-free extracts by 2-chloro-2'-deoxyadenosine 5'-triphosphate and cytochrome c

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De novo acute B cell leukemia/lymphoma with t(14;18)

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Characterisation of BCL2-JH rearrangements in follicular lymphoma: PCR detection of 3' BCL2 breakpoints and evidence of a new cluster

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Clinical significance of CD95, Bcl-2 and Bax expression and CD95 function in adult de novo acute myeloid leukemia in context of P-glycoprotein function, maturation stage, and cytogenetics.

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Bax directly induces release of cytochrome c from isolated mitochondria

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Mitochondrial control of cell death

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1 reference

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In vitro evaluation of B-CLL cells apoptotic responses to irradiation.

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Development of cyclin-dependent kinase modulators as novel therapeutic approaches for hematological malignancies

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Identifiers

10.1038/SJ.LEU.2402539

1 reference

Europe PubMed Central

PubMed publication ID

23 July 2017

Dimensions Publication ID

0 references

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

23 July 2017

ResearchGate publication ID

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