Muramyldipeptide augments the actions of lipopolysaccharide in mice by stimulating macrophages to produce pro-IL-1β and by down-regulation of the suppressor of cytokine signaling 1 (SOCS1). (Q39777644)

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English	Muramyldipeptide augments the actions of lipopolysaccharide in mice by stimulating macrophages to produce pro-IL-1β and by down-regulation of the suppressor of cytokine signaling 1 (SOCS1).	scientific article

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Europe PubMed Central

PubMed publication ID

14 September 2017

Muramyldipeptide augments the actions of lipopolysaccharide in mice by stimulating macrophages to produce pro-IL-1β and by down-regulation of the suppressor of cytokine signaling 1 (SOCS1). (English)

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14 September 2017

lipopolysaccharide

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Yoichiro Iwakura

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Yoichiro Iwakura

4

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author name string

Yosuke Shikama

1

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Europe PubMed Central

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14 September 2017

Toshinobu Kuroishi

2

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14 September 2017

Yasuhiro Nagai

3

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14 September 2017

Hidetoshi Shimauchi

5

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14 September 2017

Haruhiko Takada

6

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14 September 2017

Shunji Sugawara

7

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14 September 2017

Yasuo Endo

8

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14 September 2017

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6 November 2009

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14 September 2017

Innate Immunity

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14 September 2017

17

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14 September 2017

1

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14 September 2017

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14 September 2017

Pathogen recognition and innate immunity

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NOD-LRR proteins: role in host-microbial interactions and inflammatory disease

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Critical role of toll-like receptors and nucleotide oligomerisation domain in the regulation of health and disease

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Nod2 is a general sensor of peptidoglycan through muramyl dipeptide (MDP) detection

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Host recognition of bacterial muramyl dipeptide mediated through NOD2. Implications for Crohn's disease

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Interferon gamma receptor deficient mice are resistant to endotoxic shock

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Muramyl dipeptide enhances osteoclast formation induced by lipopolysaccharide, IL-1 alpha, and TNF-alpha through nucleotide-binding oligomerization domain 2-mediated signaling in osteoblasts

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RICK/RIP2 Mediates Innate Immune Responses Induced through Nod1 and Nod2 but Not TLRs

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NOD2 plays an important role in the inflammatory responses of microglia and astrocytes to bacterial CNS pathogens

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SOCS proteins, cytokine signalling and immune regulation

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SOCS1: a potent and multifaceted regulator of cytokines and cell-mediated inflammation.

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The role of suppressors of cytokine signalling in thymopoiesis and T cell activation

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Regulation of the immune system by SOCS family adaptor proteins

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Negative regulation of cytokine and TLR signalings by SOCS and others

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Suppressors of cytokine signaling and immunity

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Suppressor of cytokine signaling-1 regulates inflammatory bowel disease in which both IFNgamma and IL-4 are involved.

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Re-examination of the role of suppressor of cytokine signaling 1 (SOCS1) in the regulation of toll-like receptor signaling

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Pannexin-1-Mediated Recognition of Bacterial Molecules Activates the Cryopyrin Inflammasome Independent of Toll-like Receptor Signaling

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Liposome mediated depletion of macrophages: mechanism of action, preparation of liposomes and applications

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Involvement of Kupffer cells in lipopolysaccharide-induced rapid accumulation of platelets in the liver and the ensuing anaphylaxis-like shock in mice

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Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4

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Expression of the macrophage-specific antigen F4/80 during differentiation of mouse bone marrow cells in culture

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IgG-blocking antibodies inhibit IgE-mediated anaphylaxis in vivo through both antigen interception and Fc gamma RIIb cross-linking

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Engagement of NOD2 has a dual effect on proIL-1beta mRNA transcription and secretion of bioactive IL-1beta

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Synergistic stimulation of human monocytes and dendritic cells by Toll-like receptor 4 and NOD1- and NOD2-activating agonists

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Lipoteichoic acid and muramyl dipeptide synergistically induce maturation of human dendritic cells and concurrent expression of proinflammatory cytokines.

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Synergistic effect of muramyldipeptide with lipopolysaccharide or lipoteichoic acid to induce inflammatory cytokines in human monocytic cells in culture

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Proinflammatory cytokines and sepsis syndrome: not enough, or too much of a good thing?

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MDP-induced interleukin-1beta processing requires Nod2 and CIAS1/NALP3.

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A NOD2-NALP1 complex mediates caspase-1-dependent IL-1beta secretion in response to Bacillus anthracis infection and muramyl dipeptide

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Nod2-dependent regulation of innate and adaptive immunity in the intestinal tract

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Identification of bacterial muramyl dipeptide as activator of the NALP3/cryopyrin inflammasome.

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10.1177/1753425909347508

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

ResearchGate publication ID

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