Yondelis® (ET-743, Trabectedin) sensitizes cancer cell lines to CD95-mediated cell death: New molecular insight into the mechanism of action (Q39582178)

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scientific article published on May 11, 2011

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English	Yondelis® (ET-743, Trabectedin) sensitizes cancer cell lines to CD95-mediated cell death: New molecular insight into the mechanism of action	scientific article published on May 11, 2011

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scholarly article

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Europe PubMed Central

PubMed publication ID

13 September 2017

Yondelis® (ET-743, Trabectedin) sensitizes cancer cell lines to CD95-mediated cell death: new molecular insight into the mechanism of action (English)

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Europe PubMed Central

PubMed publication ID

13 September 2017

Yondelis® (ET-743, Trabectedin) sensitizes cancer cell lines to CD95-mediated cell death: New molecular insight into the mechanism of action (English)

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10.1016/J.EJPHAR.2011.02.035

https://api.crossref.org/works/10.1016/J.EJPHAR.2011.02.035

26 November 2024

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Jordi Martínez-Serra

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13 September 2017

Elena Maffiotte

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13 September 2017

Javier Martín

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13 September 2017

Teresa Bex

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Europe PubMed Central

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13 September 2017

María Navarro-Palou

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13 September 2017

Teresa Ros

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13 September 2017

Oliver Vögler

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13 September 2017

Antonio Gutiérrez

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Europe PubMed Central

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13 September 2017

Juan Carlos Amat

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13 September 2017

Rafael Ramos

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13 September 2017

Carlos Saus

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13 September 2017

Jordi Ginés

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13 September 2017

Regina Alemany

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13 September 2017

Matias Diaz

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13 September 2017

Joan Besalduch

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13 September 2017

Joana M. Plazas

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?resulttype=core&format=json&query=SRC%3AMED%20AND%20EXT_ID%3A21371453

26 November 2024

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publication date

1 March 2011

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13 September 2017

11 May 2011

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National Center for Biotechnology Information

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=21371453

26 November 2024

full work available at URL

https://api.elsevier.com/content/article/PII:S0014299911002202?httpAccept=text/xml

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10.1016/J.EJPHAR.2011.02.035

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https://api.elsevier.com/content/article/PII:S0014299911002202?httpAccept=text/plain

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10.1016/J.EJPHAR.2011.02.035

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European Journal of Pharmacology

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13 September 2017

658

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13 September 2017

2-3

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13 September 2017

57-64

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13 September 2017

Anti-inflammatory properties of the novel antitumor agent yondelis (trabectedin): inhibition of macrophage differentiation and cytokine production

1

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10.1016/J.EJPHAR.2011.02.035

https://api.crossref.org/works/10.1016/J.EJPHAR.2011.02.035

26 November 2024

Hepatotoxicity and metabolism of trabectedin: a literature review.

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10.1016/J.EJPHAR.2011.02.035

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Chemotherapy-induced apoptosis of S-type neuroblastoma cells requires caspase-9 and is augmented by CD95/Fas stimulation

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Activation of caspases-8 and -10 by FLIP(L).

4

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10.1016/J.EJPHAR.2011.02.035

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Dexamethasone enhances cell resistance to chemotherapy by increasing adhesion to extracellular matrix in human ovarian cancer cells.

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Unique pattern of ET-743 activity in different cellular systems with defined deficiencies in DNA-repair pathways

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Complete protection by high-dose dexamethasone against the hepatotoxicity of the novel antitumor drug yondelis (ET-743) in the rat

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Ecteinascidin-743 (ET-743), a natural marine compound, with a unique mechanism of action.

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ET-743: a novel agent with activity in soft-tissue sarcomas

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Differential cytostatic and apoptotic effects of ecteinascidin-743 in cancer cells. Transcription-dependent cell cycle arrest and transcription-independent JNK and mitochondrial mediated apoptosis.

10

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Akt activity protects rheumatoid synovial fibroblasts from Fas-induced apoptosis by inhibition of Bid cleavage

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Antitumor and anti-inflammatory effects of trabectedin on human myxoid liposarcoma cells.

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Apoptosis and expression of Fas/Fas ligand mRNA in bleomycin-induced pulmonary fibrosis in mice

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Camptothecin and Fas receptor agonists synergistically induce medulloblastoma cell death: ROS-dependent mechanisms

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The repression of E2F-1 is critical for the activity of Minerval against cancer.

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The long form of FLIP is an activator of caspase-8 at the Fas death-inducing signaling complex

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Interference of transcriptional activation by the antineoplastic drug ecteinascidin-743.

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p53 activates the CD95 (APO-1/Fas) gene in response to DNA damage by anticancer drugs

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Dexamethasone protects primary cultured hepatocytes from death receptor-mediated apoptosis by upregulation of cFLIP

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Canstatin inhibits Akt activation and induces Fas-dependent apoptosis in endothelial cells

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Phosphatidylinositol 3-kinase/Akt activity regulates c-FLIP expression in tumor cells

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Cellular FLICE-like inhibitory protein (C-FLIP): a novel target for cancer therapy

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Replication and homologous recombination repair regulate DNA double-strand break formation by the antitumor alkylator ecteinascidin 743.

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Ecteinascidin 743 induces protein-linked DNA breaks in human colon carcinoma HCT116 cells and is cytotoxic independently of topoisomerase I expression

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Antiproliferative activity of ecteinascidin 743 is dependent upon transcription-coupled nucleotide-excision repair.

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Role of homologous recombination in trabectedin-induced DNA damage

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Ecteinascidin 743: a minor groove alkylator that bends DNA toward the major groove

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Identifiers

10.1016/J.EJPHAR.2011.02.035

1 reference

Europe PubMed Central

PubMed publication ID

13 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

13 September 2017

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