NFκB and HIF display synergistic behaviour during hypoxic inflammation. (Q39446203)

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16 January 2020

title

NFκB and HIF display synergistic behaviour during hypoxic inflammation (English)

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16 January 2020

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16 January 2020

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16 January 2020

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16 January 2020

Ulrike Bruning

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16 January 2020

Susan F Fitzpatrick

series ordinal

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16 January 2020

Till Frank

series ordinal

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16 January 2020

publication date

9 November 2011

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Cellular and Molecular Life Sciences

16 January 2020

published in

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volume

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issue

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page(s)

1319-1329

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Regulation of physiological responses to continuous and intermittent hypoxia by hypoxia-inducible factor 1.

16 January 2020

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An intact canonical NF-κB pathway is required for inflammatory gene expression in response to hypoxia.

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The hematopoietic stem cell niche: low in oxygen but a nice place to be.

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Hypoxia up-regulates hypoxia-inducible factor-1alpha transcription by involving phosphatidylinositol 3-kinase and nuclear factor kappaB in pulmonary artery smooth muscle cells

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Reactive oxygen species activate the HIF-1alpha promoter via a functional NFkappaB site

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NF-kappaB links innate immunity to the hypoxic response through transcriptional regulation of HIF-1alpha

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Activity of hypoxia-inducible factor 2alpha is regulated by association with the NF-kappaB essential modulator

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Hypoxia-induced neutrophil survival is mediated by HIF-1alpha-dependent NF-kappaB activity

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NF-κB links CO2 sensing to innate immunity and inflammation in mammalian cells

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Small ubiquitin-related modifier (SUMO)-1 promotes glycolysis in hypoxia

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12 December 2020

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16 January 2020

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