Proteomic and transcriptomic analysis of heart failure due to volume overload in a rat aorto-caval fistula model provides support for new potential therapeutic targets - monoamine oxidase A and transglutaminase 2. (Q39415494)

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scientific article published on 11 November 2011

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English	Proteomic and transcriptomic analysis of heart failure due to volume overload in a rat aorto-caval fistula model provides support for new potential therapeutic targets - monoamine oxidase A and transglutaminase 2.	scientific article published on 11 November 2011

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16 January 2020

Proteomic and transcriptomic analysis of heart failure due to volume overload in a rat aorto-caval fistula model provides support for new potential therapeutic targets - monoamine oxidase A and transglutaminase 2 (English)

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Vojtěch Melenovský

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Jana Pospisilova

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16 January 2020

Miroslava Sedinova

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Lucie Lorkova

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Hynek Strnad

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16 January 2020

Jan Benes

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16 January 2020

Ludek Cervenka

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11 November 2011

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16 January 2020

Proteome Science

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16 January 2020

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16 January 2020

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16 January 2020

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16 January 2020

LRP16 Integrates into NF-κB Transcriptional Complex and Is Required for Its Functional Activation

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Annexin-A5 assembled into two-dimensional arrays promotes cell membrane repair

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Monoamine oxidases (MAO) in the pathogenesis of heart failure and ischemia/reperfusion injury

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Differential cardiac remodeling in preload versus afterload

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Diminished NADPH transhydrogenase activity and mitochondrial redox regulation in human failing myocardium

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11 September 2017

Transglutaminase 2: a multi-functional protein in multiple subcellular compartments

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Monoamine oxidase A-mediated enhanced catabolism of norepinephrine contributes to adverse remodeling and pump failure in hearts with pressure overload

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Involvement of tissue transglutaminase in endothelin 1-induced hypertrophy in cultured neonatal rat cardiomyocytes

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Adiponectin and cardiovascular disease

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11 September 2017

Tackling heart failure in the twenty-first century

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Altered cardiac myocyte Ca regulation in heart failure.

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Altered creatine kinase adenosine triphosphate kinetics in failing hypertrophied human myocardium

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Myocardial substrate metabolism in the normal and failing heart

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Oxygen, oxidative stress, hypoxia, and heart failure

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Annexins and Ca2+ handling in the heart

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11 September 2017

Is the failing heart energy starved? On using chemical energy to support cardiac function

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Tissue transglutaminase 2 promotes apoptosis of rat neonatal cardiomyocytes under oxidative stress

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ESC guidelines for the diagnosis and treatment of acute and chronic heart failure 2008: the Task Force for the diagnosis and treatment of acute and chronic heart failure 2008 of the European Society of Cardiology

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21 August 2018

Defective intracellular Ca2+ homeostasis contributes to myocyte dysfunction during ventricular remodelling induced by chronic volume overload in rats

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1 reference

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Metabolic characterization of volume overload heart failure due to aorto-caval fistula in rats

1 reference

https://pubmed.ncbi.nlm.nih.gov/22078724

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Relation of frequency and severity of mitral regurgitation to survival among patients with left ventricular systolic dysfunction and heart failure

1 reference

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Characterization of proteomic changes in cardiac mitochondria in streptozotocin-diabetic rats using iTRAQ™ isobaric tags

1 reference

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Myocardial morphological characteristics and proarrhythmic substrate in the rat model of heart failure due to chronic volume overload

1 reference

https://pubmed.ncbi.nlm.nih.gov/22078724

12 December 2020

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Cardiac specific overexpression of transglutaminase II (G(h)) results in a unique hypertrophy phenotype independent of phospholipase C activation

1 reference

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1 reference

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1 reference

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10.1186/1477-5956-9-69

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16 January 2020

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16 January 2020

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16 January 2020

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