MicroRNA-23a modulates tumor necrosis factor-alpha-induced osteoblasts apoptosis by directly targeting Fas. (Q39134481)

9 September 2017

title

MicroRNA-23a modulates tumor necrosis factor-alpha-induced osteoblasts apoptosis by directly targeting Fas. (English)

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9 September 2017

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Jun Dong

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9 September 2017

Xingang Cui

series ordinal

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9 September 2017

Zhensong Jiang

series ordinal

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9 September 2017

Jianmin Sun

series ordinal

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9 September 2017

publication date

1 December 2013

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Journal of Cellular Biochemistry

9 September 2017

published in

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9 September 2017

volume

114

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9 September 2017

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9 September 2017

page(s)

2738-2745

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The functions of animal microRNAs

9 September 2017

cites work

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Expression and function of TNF-family proteins and receptors in human osteoblasts.

21 January 2018

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Gene expression profiling indicate role of ER stress in miR-23a~27a~24-2 cluster induced apoptosis in HEK293T cells

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Apoptosis: a review of programmed cell death

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Extrinsic versus intrinsic apoptosis pathways in anticancer chemotherapy

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MicroRNA-17-92a upregulation by estrogen leads to Bim targeting and inhibition of osteoblast apoptosis

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The grapevine genome sequence suggests ancestral hexaploidization in major angiosperm phyla

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Combined treatment with verrucarin A and tumor necrosis factor-α sensitizes apoptosis by overexpression of nuclear factor-kappaB-mediated Fas.

21 January 2018

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Osteoblast programmed cell death (apoptosis): modulation by growth factors and cytokines

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Effect of TNF-alpha on Raji cells at different cellular levels estimated by various methods

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The concentration of TNF-alpha correlate with number of inflammatory cells and degree of vascularization in radicular cysts

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Elevated TNF-alpha and LDH without parathormone disturbance is associated with diffuse osteolytic lesions in leukemic transformation of myelofibrosis

21 January 2018

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Expression profiling of microRNAs in RAW264.7 cells treated with a combination of tumor necrosis factor alpha and RANKL during osteoclast differentiation

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Fas and Fas ligand interaction is necessary for human osteoblast apoptosis.

21 January 2018

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Transforming growth factor-beta 1 induces apoptosis through Fas ligand-independent activation of the Fas death pathway in human gastric SNU-620 carcinoma cells.

21 January 2018

1 reference

Physiological functions of tumor necrosis factor and the consequences of its pathologic overexpression or blockade: mouse models

21 January 2018

1 reference

Redundant miR-3077-5p and miR-705 mediate the shift of mesenchymal stem cell lineage commitment to adipocyte in osteoporosis bone marrow

21 January 2018

1 reference

Functional investigation of Fas ligand expressions in human non-small cell lung cancer cells and its clinical implications

21 January 2018

1 reference

Induction of osteoclastogenesis in an in vitro model of Gaucher disease is mediated by T cells via TNF-α.

21 January 2018

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The transmembrane domains of TNF-related apoptosis-inducing ligand (TRAIL) receptors 1 and 2 co-regulate apoptotic signaling capacity

21 January 2018

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Identification of MicroRNAs Involved in Hypoxia- and Serum Deprivation-Induced Apoptosis in Mesenchymal Stem Cells

21 January 2018

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Differential expression of the Fas-Fas ligand system on cytokine-induced apoptotic cell death in mouse osteoblastic cells

21 January 2018

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Effects of down-regulation of microRNA-23a on TNF-α-induced endothelial cell apoptosis through caspase-dependent pathways.

21 January 2018

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Inhibition of osteoblastogenesis and promotion of apoptosis of osteoblasts and osteocytes by glucocorticoids. Potential mechanisms of their deleterious effects on bone

21 January 2018

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miR-155 modulates TNF-α-inhibited osteogenic differentiation by targeting SOCS1 expression

21 January 2018

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The muscle-specific microRNA miR-1 regulates cardiac arrhythmogenic potential by targeting GJA1 and KCNJ2

21 January 2018

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Tumor necrosis factor-alpha (TNF) stimulates RANKL-induced osteoclastogenesis via coupling of TNF type 1 receptor and RANK signaling pathways

21 January 2018

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21 January 2018

Identifiers

DOI

10.1002/JCB.24622

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9 September 2017

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