Metformin attenuates graft-versus-host disease via restricting mammalian target of rapamycin/signal transducer and activator of transcription 3 and promoting adenosine monophosphate-activated protein kinase-autophagy for the balance between T helper (Q38415035)

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scientific article published on 22 March 2016

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English	Metformin attenuates graft-versus-host disease via restricting mammalian target of rapamycin/signal transducer and activator of transcription 3 and promoting adenosine monophosphate-activated protein kinase-autophagy for the balance between T helper	scientific article published on 22 March 2016

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scholarly article

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2 September 2017

Metformin attenuates graft-versus-host disease via restricting mammalian target of rapamycin/signal transducer and activator of transcription 3 and promoting adenosine monophosphate-activated protein kinase-autophagy for the balance between T helper (English)

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2 September 2017

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Min-Jung Park

1

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2 September 2017

Seon-Yeong Lee

2

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2 September 2017

Su-Jin Moon

3

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2 September 2017

Hye-Jin Son

4

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2 September 2017

Sung-Hee Lee

5

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2 September 2017

Eun-Kyung Kim

6

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2 September 2017

Jae-Kyeong Byun

7

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2 September 2017

Dong Yun Shin

8

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2 September 2017

Sung-Hwan Park

9

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2 September 2017

Chul-Woo Yang

10

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2 September 2017

Mi-La Cho

11

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2 September 2017

publication date

22 March 2016

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2 September 2017

Translational Research

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2 September 2017

173

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2 September 2017

115-130

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2 September 2017

Graft-versus-host disease

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Metabolic pathways in T cell fate and function

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Fuel feeds function: energy metabolism and the T-cell response

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AMP-activated protein kinase: ancient energy gauge provides clues to modern understanding of metabolism

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Role of AMP-activated protein kinase in mechanism of metformin action

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AMP-activated/SNF1 protein kinases: conserved guardians of cellular energy

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AMP-activated protein kinase suppresses protein synthesis in rat skeletal muscle through down-regulated mammalian target of rapamycin (mTOR) signaling

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Regulation and function of mTOR signalling in T cell fate decisions

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mTOR‐STAT3‐notch signalling contributes to ALDH2‐induced protection against cardiac contractile dysfunction and autophagy under alcoholism

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Serine phosphorylation and maximal activation of STAT3 during CNTF signaling is mediated by the rapamycin target mTOR

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Therapeutic metformin/AMPK activation blocked lymphoma cell growth via inhibition of mTOR pathway and induction of autophagy

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Prevention of graft-versus-host disease by intra-bone marrow injection of donor T cells

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TOS motif-mediated raptor binding regulates 4E-BP1 multisite phosphorylation and function

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The mammalian target of rapamycin (mTOR) partner, raptor, binds the mTOR substrates p70 S6 kinase and 4E-BP1 through their TOR signaling (TOS) motif

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Hypoxia signalling through mTOR and the unfolded protein response in cancer

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The oxidative status of blood cells in a murine model of graft-versus-host disease

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The Nrf2-antioxidant response element signaling pathway and its activation by oxidative stress

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Identification of novel modulators of mitochondrial function by a genome-wide RNAi screen in Drosophila melanogaster

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AMP-activated protein kinase induces a p53-dependent metabolic checkpoint

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p53 and autophagy in cancer: Guardian of the genome meets guardian of the proteome

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Regulatory T Cells and IL-17-Producing Cells in Graft-Versus-Host Disease

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Metformin targets Stat3 to inhibit cell growth and induce apoptosis in triple-negative breast cancers

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Human CD8+ regulatory T cells inhibit GVHD and preserve general immunity in humanized mice

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The coordinate regulation of the p53 and mTOR pathways in cells

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Cellular and molecular mechanisms of metformin: an overview

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Hypertension and diabetes mellitus in adult and pediatric survivors of allogeneic hematopoietic cell transplantation

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AMP-activated protein kinase inhibits IL-6-stimulated inflammatory response in human liver cells by suppressing phosphorylation of signal transducer and activator of transcription 3 (STAT3).

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Effects of metformin on retinoblastoma growth in vitro and in vivo.

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Cutting edge: distinct glycolytic and lipid oxidative metabolic programs are essential for effector and regulatory CD4+ T cell subsets.

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The AMPK signalling pathway coordinates cell growth, autophagy and metabolism

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CD4+ and CD8+ regulatory T cells generated ex vivo with IL-2 and TGF-beta suppress a stimulatory graft-versus-host disease with a lupus-like syndrome

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Identifiers

10.1016/J.TRSL.2016.03.006

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

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