Comparative proteomics profiling of a phospholamban mutant mouse model of dilated cardiomyopathy reveals progressive intracellular stress responses (Q38295801)

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English	Comparative proteomics profiling of a phospholamban mutant mouse model of dilated cardiomyopathy reveals progressive intracellular stress responses	scientific article

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1 September 2017

Comparative proteomics profiling of a phospholamban mutant mouse model of dilated cardiomyopathy reveals progressive intracellular stress responses (English)

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dilated cardiomyopathy

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based on heuristic

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Desmond G. Higgins

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Parveen Sharma

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Thomas Kislinger

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Thomas Kislinger

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Andrew Emili

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Ruth Isserlin

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Christine E. Seidman

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Christine E Seidman

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Brendan J. Frey

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Brendan Frey

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Anthony O Gramolini

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Rasoul Alikhani-Koopaei

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Vincent Fong

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Natalie J Thompson

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Gavin Y Oudit

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Maria G Trivieri

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Ailís Fagan

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Anitha Kannan

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Hendrik Huedig

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George Hess

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Sara Arab

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Jonathan G Seidman

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Marc Perry

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Peter H Backx

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Peter P Liu

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David H MacLennan

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publication date

30 November 2007

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Molecular & Cellular Proteomics

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519-533

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Heart disease and stroke statistics--2006 update: a report from the American Heart Association Statistics Committee and Stroke Statistics Subcommittee

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A calcineurin-dependent transcriptional pathway for cardiac hypertrophy

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Learning from failure: congestive heart failure in the postgenomic age

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Dilated cardiomyopathy and heart failure caused by a mutation in phospholamban

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The transgenic expression of highly inhibitory monomeric forms of phospholamban in mouse heart impairs cardiac contractility

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Regulation of myocardial contractility and cell size by distinct PI3K-PTEN signaling pathways

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Angiotensin-converting enzyme 2 is an essential regulator of heart function

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Taurine supplementation reduces oxidative stress and improves cardiovascular function in an iron-overload murine model

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Cardiac-specific overexpression of sarcolipin in phospholamban null mice impairs myocyte function that is restored by phosphorylation

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Cardiac-specific elevations in thyroid hormone enhance contractility and prevent pressure overload-induced cardiac dysfunction

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PRISM, a generic large scale proteomic investigation strategy for mammals

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Expression of the utrophin gene during myogenic differentiation

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PSORT: a program for detecting sorting signals in proteins and predicting their subcellular localization

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MAPU: Max-Planck Unified database of organellar, cellular, tissue and body fluid proteomes

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Hypertrophy, pathology, and molecular markers of cardiac pathogenesis.

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Alterations in mouse cardiac proteome after in vivo myocardial infarction: permanent ischaemia versus ischaemia-reperfusion

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MLP-deficient mice exhibit a disruption of cardiac cytoarchitectural organization, dilated cardiomyopathy, and heart failure

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Stress pathways and heart failure

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Prolonged endoplasmic reticulum stress in hypertrophic and failing heart after aortic constriction: possible contribution of endoplasmic reticulum stress to cardiac myocyte apoptosis

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Endoplasmic reticulum stress: cell life and death decisions.

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Dilated cardiomyopathy caused by aberrant endoplasmic reticulum quality control in mutant KDEL receptor transgenic mice

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10.1074/MCP.M700245-MCP200

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Europe PubMed Central

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1 September 2017

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1 September 2017

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