IKK/NF-kappaB signaling in intestinal epithelial cells controls immune homeostasis in the gut. (Q37349789)

19 August 2017

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title

IKK/NF-kappaB signaling in intestinal epithelial cells controls immune homeostasis in the gut. (English)

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19 August 2017

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M Pasparakis

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1 November 2008

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19 August 2017

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19 August 2017

volume

1 Suppl 1

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19 August 2017

page(s)

S54-7

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The fundamental basis of inflammatory bowel disease

19 August 2017

cites work

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The IKK complex: an integrator of all signals that activate NF-kappaB?

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Rel/NF-kappa B/I kappa B family: intimate tales of association and dissociation

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Predominant Role of NF-κB p65 in the Pathogenesis of Chronic Intestinal Inflammation

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Activation of nuclear factor kappa B inflammatory bowel disease

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A NFKB1 promoter polymorphism is involved in susceptibility to ulcerative colitis.

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Local administration of antisense phosphorothioate oligonucleotides to the p65 subunit of NF-kappa B abrogates established experimental colitis in mice

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Epithelial NEMO links innate immunity to chronic intestinal inflammation

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The two NF-kappaB activation pathways and their role in innate and adaptive immunity

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Integrating cell-signalling pathways with NF-kappaB and IKK function

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NEMO/IKK gamma-deficient mice model incontinentia pigmenti.

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Cis elements of the villin gene control expression in restricted domains of the vertical (crypt) and horizontal (duodenum, cecum) axes of the intestine.

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A chromosome 8 gene-cluster polymorphism with low human beta-defensin 2 gene copy number predisposes to Crohn disease of the colon

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NF-kappaB-related genetic diseases

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Pediatric inflammatory bowel disease: what children can teach adults

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Identifiers

DOI

10.1038/MI.2008.53

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19 August 2017

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