O-GlcNAcylation-inducing treatments inhibit estrogen receptor α expression and confer resistance to 4-OH-tamoxifen in human breast cancer-derived MCF-7 cells. (Q37062403)

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scientific article published on 11 July 2013

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English	O-GlcNAcylation-inducing treatments inhibit estrogen receptor α expression and confer resistance to 4-OH-tamoxifen in human breast cancer-derived MCF-7 cells.	scientific article published on 11 July 2013

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7 March 2020

O-GlcNAcylation-inducing treatments inhibit estrogen receptor α expression and confer resistance to 4-OH-tamoxifen in human breast cancer-derived MCF-7 cells (English)

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7 March 2020

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7 March 2020

Christiane S Hampe

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7 March 2020

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7 March 2020

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Shahzina Kanwal

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7 March 2020

Yann Fardini

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7 March 2020

Patrick Pagesy

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7 March 2020

Thierry N'tumba-Byn

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7 March 2020

Cécile Pierre-Eugène

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11 July 2013

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7 March 2020

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7 March 2020

e69150

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7 March 2020

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Creative Commons Attribution 4.0 International

11 July 2013

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The hexosamine biosynthetic pathway and O-GlcNAcylation drive the expression of β-catenin and cell proliferation

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Gene expression of O-GlcNAc cycling enzymes in human breast cancers

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O-GlcNAcylation plays a role in tumor recurrence of hepatocellular carcinoma following liver transplantation

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O-GlcNAcylation is a novel regulator of lung and colon cancer malignancy

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Elevated O-linked N-acetylglucosamine correlated with reduced Sp1 cooperative DNA binding with its collaborating factors in vivo

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GlcNAcylation plays an essential role in breast cancer metastasis

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29 September 2017

Nutrient sensor O-GlcNAc transferase regulates breast cancer tumorigenesis through targeting of the oncogenic transcription factor FoxM1.

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O-GlcNAc inhibits interaction between Sp1 and sterol regulatory element binding protein 2.

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29 September 2017

c-Jun activation is required for 4-hydroxytamoxifen-induced cell death in breast cancer cells.

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29 September 2017

Dysregulation of the nutrient/stress sensor O-GlcNAcylation is involved in the etiology of cardiovascular disorders, type-2 diabetes and Alzheimer's disease.

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29 September 2017

Implication of protein tyrosine phosphatase 1B in MCF-7 cell proliferation and resistance to 4-OH tamoxifen

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29 September 2017

O-GlcNAcylation of Sp1 interrupts Sp1 interaction with NF-Y.

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29 September 2017

O-GlcNAc inhibits interaction between Sp1 and Elf-1 transcription factors

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29 September 2017

O-GlcNAc modification of Sp1 inhibits the functional interaction between Sp1 and Oct1

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29 September 2017

Nutrition, metabolic factors and cancer risk

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O-GlcNAc modification of transcription factors, glucose sensing and glucotoxicity

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29 September 2017

A candidate molecular signature associated with tamoxifen failure in primary breast cancer

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Rational management of endocrine resistance in breast cancer: a comprehensive review of estrogen receptor biology, treatment options, and future directions.

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Glucose avidity of carcinomas.

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29 September 2017

Regulation of Akt signaling by O-GlcNAc in euglycemia.

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Characterization of IRA/IRB hybrid insulin receptors using bioluminescence resonance energy transfer

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29 September 2017

Insulin resistance, obesity and breast cancer risk

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29 September 2017

O-GlcNAc modification of FoxO1 increases its transcriptional activity: a role in the glucotoxicity phenomenon?

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29 September 2017

Phosphoinositide signalling links O-GlcNAc transferase to insulin resistance

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O-glycosylation of FoxO1 increases its transcriptional activity towards the glucose 6-phosphatase gene

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Cycling of O-linked beta-N-acetylglucosamine on nucleocytoplasmic proteins

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Interaction with Grb14 results in site-specific regulation of tyrosine phosphorylation of the insulin receptor

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The value of estrogen and progesterone receptors in the treatment of breast cancer

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Receptor levels in breast cancer

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Decreased Egr-1 expression in human, mouse and rat mammary cells and tissues correlates with tumor formation

1 reference

https://pubmed.ncbi.nlm.nih.gov/23935944

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inferred from PubMed ID database lookup

Energy availability and mammary carcinogenesis: effects of calorie restriction and exercise

1 reference

https://pubmed.ncbi.nlm.nih.gov/23935944

12 December 2020

based on heuristic

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Fibroblast growth factor-2 has opposite effects on human breast cancer MCF-7 cell growth depending on the activation level of the mitogen-activated protein kinase pathway

1 reference

https://pubmed.ncbi.nlm.nih.gov/23935944

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Early growth response gene 1 (EGR1) is deleted in estrogen receptor-negative human breast carcinoma

1 reference

https://pubmed.ncbi.nlm.nih.gov/23935944

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

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10.1371/JOURNAL.PONE.0069150

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7 March 2020

2013PLoSO...869150K

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7 March 2020

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7 March 2020

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