B cells induce tolerance by presenting endogenous peptide-IgG on MHC class II molecules via an IFN-gamma-inducible lysosomal thiol reductase-dependent pathway (Q36983280)

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scientific article published on July 2008

Language	Label	Description	Also known as
English	B cells induce tolerance by presenting endogenous peptide-IgG on MHC class II molecules via an IFN-gamma-inducible lysosomal thiol reductase-dependent pathway	scientific article published on July 2008

Statements

scholarly article

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

B cells induce tolerance by presenting endogenous peptide-IgG on MHC class II molecules via an IFN-gamma-inducible lysosomal thiol reductase-dependent pathway (English)

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

author name string

Yan Su

1

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

Gregory Carey

2

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

Maja Maric

3

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

David W Scott

4

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

language of work or name

0 references

publication date

1 July 2008

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Europe PubMed Central

PMC publication ID

17 August 2017

number of pages

8

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based on heuristic

inferred from page(s)

Journal of Immunology

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

181

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

2

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

1153-1160

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

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1 reference

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Functional requirements for the lysosomal thiol reductase GILT in MHC class II-restricted antigen processing

1 reference

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1 reference

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1 reference

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Central tolerance: learning self-control in the thymus

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Gene transfer of Ig-fusion proteins into B cells prevents and treats autoimmune diseases

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Gene therapeutic approaches to induction and maintenance of tolerance

1 reference

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Defective antigen processing in GILT-free mice

1 reference

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Protein translocation across membranes

1 reference

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Retroviral gene therapy with an immunoglobulin-antigen fusion construct protects from experimental autoimmune uveitis

1 reference

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Cytoplasmic processing is a prerequisite for presentation of an endogenous antigen by major histocompatibility complex class II proteins

1 reference

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Enzymatic reduction of disulfide bonds in lysosomes: characterization of a gamma-interferon-inducible lysosomal thiol reductase (GILT)

1 reference

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1 reference

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An intralysosomal hsp70 is required for a selective pathway of lysosomal protein degradation

1 reference

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Presentation of a cytosolic antigen by major histocompatibility complex class II molecules requires a long-lived form of the antigen

1 reference

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Epitope-specific tolerance induction with an engineered immunoglobulin

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Specificity and promiscuity among naturally processed peptides bound to HLA-DR alleles

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HLA class II-restricted presentation of cytoplasmic measles virus antigens to cytotoxic T cells

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Small B cells as antigen-presenting cells in the induction of tolerance to soluble protein antigens

1 reference

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Sequence analysis of peptides bound to MHC class II molecules

1 reference

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Persistence of antigen-binding cells with surface tolerogen: isologous versus heterologous immunoglobulin carriers

1 reference

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B cells turn off virgin but not memory T cells

1 reference

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Bone marrow transplantation combined with gene therapy to induce antigen-specific tolerance and ameliorate EAE.

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Processing and presentation of HLA class I and II epitopes by dendritic cells after transfection with in vitro-transcribed MUC1 RNA.

1 reference

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Retroviral delivery of GAD-IgG fusion construct induces tolerance and modulates diabetes: a role for CD4+ regulatory T cells and TGF-beta?

1 reference

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A novel retroviral gene therapy approach to inhibit specific antibody production and suppress experimental autoimmune encephalomyelitis induced by MOG and MBP.

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In vivo induction of tolerance by an Ig peptide is not affected by the deletion of FcR or a mutated IgG Fc fragment

1 reference

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In vivo MHC class II presentation of cytosolic proteins revealed by rapid automated tandem mass spectrometry and functional analyses.

1 reference

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Mechanisms of tolerance induction by a gene-transferred peptide-IgG fusion protein expressed in B lineage cells

1 reference

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Tolerogenicity of resting and activated B cells

1 reference

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Tumor cells present MHC class II-restricted nuclear and mitochondrial antigens and are the predominant antigen presenting cells in vivo

1 reference

https://pubmed.ncbi.nlm.nih.gov/18606668

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Disulfide reduction in major histocompatibility complex class II-restricted antigen processing by interferon-gamma-inducible lysosomal thiol reductase

1 reference

https://pubmed.ncbi.nlm.nih.gov/18606668

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Distinct dendritic cell populations sequentially present antigen to CD4 T cells and stimulate different aspects of cell-mediated immunity

1 reference

https://pubmed.ncbi.nlm.nih.gov/18606668

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Effect on tolerance induction of the mode of attachment of the hapten the carrier

1 reference

https://pubmed.ncbi.nlm.nih.gov/18606668

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Loss of carrier-determined tolerance in vitro with loss of receptor blockade

1 reference

https://pubmed.ncbi.nlm.nih.gov/18606668

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

On the complexity of self

1 reference

https://pubmed.ncbi.nlm.nih.gov/18606668

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Both resting and activated B lymphocytes expressing engineered peptide-Ig molecules serve as highly efficient tolerogenic vehicles in immunocompetent adult recipients

1 reference

https://pubmed.ncbi.nlm.nih.gov/18606668

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.4049/JIMMUNOL.181.2.1153

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

17 August 2017

ResearchGate publication ID

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