Mechanisms of disease: multiple endocrine neoplasia type 1-relation to chromatin modifications and transcription regulation. (Q36615929)

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20 December 2019

review article

1 reference

title

Mechanisms of disease: multiple endocrine neoplasia type 1-relation to chromatin modifications and transcription regulation (English)

1 reference

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multiple endocrine neoplasia

20 December 2019

main subject

1 reference

1 reference

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20 December 2019

Jo Wm Höppener

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20 December 2019

Ht Marc Timmers

series ordinal

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20 December 2019

Cornelis Jm Lips

series ordinal

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20 December 2019

publication date

1 October 2006

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Nature Clinical Practice Endocrinology and Metabolism

20 December 2019

published in

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20 December 2019

volume

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20 December 2019

issue

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20 December 2019

page(s)

562-570

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Menin represses JunD-activated transcription by a histone deacetylase-dependent mechanism

20 December 2019

cites work

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

Positional cloning of the gene for multiple endocrine neoplasia-type 1

21 January 2018

1 reference

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7 January 2021

Current concepts in the surgical management of multiple endocrine neoplasia type 1 pancreatic-duodenal disease. Results in the treatment of 40 patients with Zollinger-Ellison syndrome, hypoglycaemia or both

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7 January 2021

Do patients with multiple endocrine neoplasia syndrome type 1 benefit from periodical screening?

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7 January 2021

Criteria for mutation analysis in MEN 1-suspected patients: MEN 1 case-finding

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7 January 2021

Osteoporosis in multiple endocrine neoplasia type 1: severity, clinical significance, relationship to primary hyperparathyroidism, and response to parathyroidectomy

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7 January 2021

Multiple endocrine neoplasia type 1 gene maps to chromosome 11 and is lost in insulinoma

1 reference

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7 January 2021

Somatic mutation of the MEN1 gene in parathyroid tumours

1 reference

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7 January 2021

A mouse model of multiple endocrine neoplasia, type 1, develops multiple endocrine tumors

1 reference

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7 January 2021

Genotype-phenotype analysis in multiple endocrine neoplasia type 1

1 reference

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7 January 2021

Germline mutation profile of MEN1 in multiple endocrine neoplasia type 1: search for correlation between phenotype and the functional domains of the MEN1 protein.

1 reference

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7 January 2021

Multiple endocrine neoplasia type 1 (MEN1) germline mutations in familial isolated primary hyperparathyroidism

1 reference

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7 January 2021

Menin, the product of the MEN1 gene, is a nuclear protein

1 reference

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7 January 2021

Functional studies of the MEN1 gene

1 reference

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7 January 2021

Functional interaction between tumor suppressor menin and activator of S-phase kinase

1 reference

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7 January 2021

Menin, a gene product responsible for multiple endocrine neoplasia type 1, interacts with the putative tumor metastasis suppressor nm23

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

The 32-kilodalton subunit of replication protein A interacts with menin, the product of the MEN1 tumor suppressor gene

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Menin's interaction with glial fibrillary acidic protein and vimentin suggests a role for the intermediate filament network in regulating menin activity

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Menin, a tumor suppressor, associates with nonmuscle myosin II-A heavy chain

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Menin interacts with the AP1 transcription factor JunD and represses JunD-activated transcription

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

The tumor suppressor protein menin interacts with NF-kappaB proteins and inhibits NF-kappaB-mediated transactivation

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Inactivation of menin, a Smad3-interacting protein, blocks transforming growth factor type beta signaling

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Menin interacts directly with the homeobox-containing protein Pem

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Menin associates with a trithorax family histone methyltransferase complex and with the hoxc8 locus

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Leukemia proto-oncoprotein MLL forms a SET1-like histone methyltransferase complex with menin to regulate Hox gene expression

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Menin uncouples Elk-1, JunD and c-Jun phosphorylation from MAP kinase activation

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Transcription factor JunD, deprived of menin, switches from growth suppressor to growth promoter

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Telomeres and human disease: ageing, cancer and beyond

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Multiple tumor suppressor pathways negatively regulate telomerase

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Menin inactivation leads to loss of transforming growth factor beta inhibition of parathyroid cell proliferation and parathyroid hormone secretion

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Activin inhibits pituitary prolactin expression and cell growth through Smads, Pit-1 and menin

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Active genes are tri-methylated at K4 of histone H3

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Global and Hox-specific roles for the MLL1 methyltransferase

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

The menin tumor suppressor protein is an essential oncogenic cofactor for MLL-associated leukemogenesis

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Functional collaboration between different cyclin-dependent kinase inhibitors suppresses tumor growth with distinct tissue specificity

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Menin and MLL cooperatively regulate expression of cyclin-dependent kinase inhibitors

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Menin regulates pancreatic islet growth by promoting histone methylation and expression of genes encoding p27Kip1 and p18INK4c

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Menin links estrogen receptor activation to histone H3K4 trimethylation

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Direct binding of DNA by tumor suppressor menin

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Loss of acetylation at Lys16 and trimethylation at Lys20 of histone H4 is a common hallmark of human cancer

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Global histone modification patterns predict risk of prostate cancer recurrence

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Menin missense mutants associated with multiple endocrine neoplasia type 1 are rapidly degraded via the ubiquitin-proteasome pathway

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Hypermutability in a Drosophila model for multiple endocrine neoplasia type 1.

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Meningiomas may be a component tumor of multiple endocrine neoplasia type 1.

1 reference

https://api.crossref.org/works/10.1038%2FNCPENDMET0292

7 January 2021

Identifiers

DOI

10.1038/NCPENDMET0292

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17024155%20AND%20SRC:MED&resulttype=core&format=json

20 December 2019

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17024155%20AND%20SRC:MED&resulttype=core&format=json