Normal adaptation of Candida albicans to the murine gastrointestinal tract requires Efg1p-dependent regulation of metabolic and host defense genes (Q36505103)

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scientific article published on 2 November 2012

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English	Normal adaptation of Candida albicans to the murine gastrointestinal tract requires Efg1p-dependent regulation of metabolic and host defense genes	scientific article published on 2 November 2012

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scholarly article

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

Normal adaptation of Candida albicans to the murine gastrointestinal tract requires Efg1p-dependent regulation of metabolic and host defense genes (English)

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

metabolic adaptation

0 references

Candida albicans

1 reference

based on heuristic

inferred from title

Malcolm Whiteway

3

object named as

Malcolm Whiteway

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

author name string

Jessica V Pierce

1

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

Daniel Dignard

2

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

Carol A Kumamoto

4

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

publication date

2 November 2012

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Europe PubMed Central

PMC publication ID

14 August 2017

Eukaryotic Cell

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Europe PubMed Central

PMC publication ID

14 August 2017

12

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

1

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

37-49

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

Variation in Candida albicans EFG1 expression enables host-dependent changes in colonizing fungal populations

1 reference

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Divergent targets of Candida albicans biofilm regulator Bcr1 in vitro and in vivo

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Eating for two: how metabolism establishes interspecies interactions in the gut

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Candida albicans cell wall glycosylation may be indirectly required for activation of epithelial cell proinflammatory responses

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An iron homeostasis regulatory circuit with reciprocal roles in Candida albicans commensalism and pathogenesis

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Intracellular acetyl unit transport in fungal carbon metabolism

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Adaptations of Candida albicans for growth in the mammalian intestinal tract

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Candida albicans Hyr1p confers resistance to neutrophil killing and is a potential vaccine target.

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Contribution of Candida albicans cell wall components to recognition by and escape from murine macrophages

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Clinical aspects of invasive candidiasis in paediatric patients

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Time course global gene expression analysis of an in vivo Candida biofilm.

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Genome-wide analysis of Candida albicans gene expression patterns during infection of the mammalian kidney.

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Responses to hypoxia in fungal pathogens

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Epidemiology of candidemia in intensive care units

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The Yak1 kinase is involved in the initiation and maintenance of hyphal growth in Candida albicans

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Mucosal damage and neutropenia are required for Candida albicans dissemination

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Carnitine-dependent transport of acetyl coenzyme A in Candida albicans is essential for growth on nonfermentable carbon sources and contributes to biofilm formation

1 reference

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Self-regulation of Candida albicans population size during GI colonization

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30 September 2017

In vivo transcript profiling of Candida albicans identifies a gene essential for interepithelial dissemination

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Developmental regulation of an adhesin gene during cellular morphogenesis in the fungal pathogen Candida albicans.

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Niche-specific regulation of central metabolic pathways in a fungal pathogen.

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The effects of HIV infection on oral mucosal immunity.

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Identification and characterization of Cor33p, a novel protein implicated in tolerance towards oxidative stress in Candida albicans

1 reference

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Induction of the Candida albicans filamentous growth program by relief of transcriptional repression: a genome-wide analysis

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30 September 2017

Susceptibility of germfree phagocyte oxidase- and nitric oxide synthase 2-deficient mice, defective in the production of reactive metabolites of both oxygen and nitrogen, to mucosal and systemic candidiasis of endogenous origin

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30 September 2017

Transcriptional profiling in Candida albicans reveals new adaptive responses to extracellular pH and functions for Rim101p

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Transcriptional response of Candida albicans upon internalization by macrophages

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APSES proteins regulate morphogenesis and metabolism in Candida albicans

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Microflora of the gastrointestinal tract: a review.

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Colonization and epithelial adhesion in the pathogenesis of neonatal candidiasis

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Superoxide Dismutases inCandida albicans: Transcriptional Regulation and Functional Characterization of the Hyphal-inducedSOD5Gene

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Engineered control of cell morphology in vivo reveals distinct roles for yeast and filamentous forms of Candida albicans during infection

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Regulation of short-chain fatty acid production

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Stress-induced gene expression in Candida albicans: absence of a general stress response.

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EFG1 is a major regulator of cell wall dynamics in Candida albicans as revealed by DNA microarrays.

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Transcription profiling of Candida albicans cells undergoing the yeast-to-hyphal transition

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Candida albicans hyphal formation and the expression of the Efg1-regulated proteinases Sap4 to Sap6 are required for the invasion of parenchymal organs

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Transcriptional control of dimorphism in Candida albicans

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30 September 2017

DNA array studies demonstrate convergent regulation of virulence factors by Cph1, Cph2, and Efg1 in Candida albicans

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Aspartyl proteinases of Candida albicans and their role in pathogenicity

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The basic helix-loop-helix transcription factor Cph2 regulates hyphal development in Candida albicans partly via TEC1

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Virulence factors of Candida albicans

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The ALS gene family of Candida albicans

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Candida albicans RIM101 pH response pathway is required for host-pathogen interactions

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TUP1, CPH1 and EFG1 make independent contributions to filamentation in candida albicans

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HWP1 functions in the morphological development of Candida albicans downstream of EFG1, TUP1, and RBF1.

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Fungal infections

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Iron deprivation inducesEFG1-mediated hyphal development inCandida albicanswithout affecting biofilm formation

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Identifiers

10.1128/EC.00236-12

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

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